Evidence map›Paper›PMID 39768142›Full record

ArticleCells2024

Adipose-Derived Mesenchymal Stem Cells (ADSCs) Have Anti-Fibrotic Effects on Lung Fibroblasts from Idiopathic Pulmonary Fibrosis (IPF) Patients.

Noriko Ouji-Sageshima, Aiko Hiyama, Makiko Kumamoto, Masahiro Kitabatake, Atsushi Hara, Ryutaro Furukawa, Shigeto Hontsu, Takeshi Kawaguchi, Noriyoshi Sawabata, Shigeo Muro and 1 more

Abstract read
In one paragraph

Article in Cells, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 5 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
5citing papers in PubMed, 1 pooled it
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

5 citing papers in PubMed, 1 synthesis or guideline pooled it.

  1. Pooled it
  2. Multi-omics insights into the mechanisms and prognosis of IPF.Genes and environment : the official journal of the Japanese Environmental Mutagen Society · 2026
    Review
  3. Article
  4. Review
  5. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

11 authors.

Noriko Ouji-SageshimaDepartment of Immunology, Nara Medical University, Kashihara 634-8521, Nara, Japan.ORCID 0009-0003-8876-4717
Aiko HiyamaDepartment of Immunology, Nara Medical University, Kashihara 634-8521, Nara, Japan.
Makiko KumamotoDepartment of Respiratory Medicine, Nara Medical University, Kashihara 634-8521, Nara, Japan.
Masahiro KitabatakeDepartment of Immunology, Nara Medical University, Kashihara 634-8521, Nara, Japan.
Atsushi HaraDepartment of Immunology, Nara Medical University, Kashihara 634-8521, Nara, Japan.
Ryutaro FurukawaDepartment of Immunology, Nara Medical University, Kashihara 634-8521, Nara, Japan.
Shigeto HontsuDepartment of Respiratory Medicine, Nara Medical University, Kashihara 634-8521, Nara, Japan.
Takeshi KawaguchiDepartment of Thoracic and Cardiovascular Surgery, Nara Medical University, Kashihara 634-8521, Nara, Japan.
Noriyoshi SawabataDepartment of Thoracic and Cardiovascular Surgery, Nara Medical University, Kashihara 634-8521, Nara, Japan.ORCID 0000-0002-4899-1005
Shigeo MuroDepartment of Respiratory Medicine, Nara Medical University, Kashihara 634-8521, Nara, Japan.ORCID 0000-0001-7452-9191
Toshihiro ItoDepartment of Immunology, Nara Medical University, Kashihara 634-8521, Nara, Japan.

Funding

Japan Society for the Promotion of Science JP16H05310Japan Society for the Promotion of Science JP16K19463Japan Society for the Promotion of Science JP17K09663Japan Society for the Promotion of Science JP19K08661Japan Society for the Promotion of Science JP21K09162Japan Society for the Promotion of Science JP24K02460ROHTO Pharmaceutical Co., Ltd. ROHTO Pharmaceutical Co., Ltd.
6 · The paper itself

Abstract

Idiopathic pulmonary fibrosis (IPF) is the most common type of fibrosis in lungs, characterized as a chronic and progressive interstitial lung disease involving pathological findings of fibrosis with a median survival of 3 years. Despite the knowledge accumulated regarding IPF from basic and clinical research, an effective medical therapy for the condition remains to be established. Thus, it is necessary for further research, including stem cell therapy, which will provide new insights into and expectations for IPF treatment. Recently, it has been reported that one of the new therapeutic candidates for IPF is adipose-derived mesenchymal stem cells (ADSCs), which have several benefits, such as easy accessibility and minimal morbidity compared to bone marrow-derived mesenchymal stem cells. Therefore, we investigated the possibility of ADSCs as a therapeutic candidate for IPF. Using human lung fibroblasts (LFs) from IPF patients, we demonstrated that human IPF LFs cocultured with ADSCs led to reduced fibrosis-related genes. Further analysis revealed that ADSCs prevented the activation of the ERK signaling pathway in IPF LFs via the upregulation of protein tyrosine phosphatase receptor-type R (PTPRR), which negatively regulates the ERK signaling pathway. Moreover, we demonstrated that intravascular administration of ADSCs improved the pathogenesis of bleomycin-induced pulmonary fibrosis with reduced collagen deposition in histology and hydroxyproline quantification and collagen markers such as the gene expression of types I and III collagen and α-smooth muscle actin (α-SMA) in a murine model. ADSC transfer was also investigated in a humanized mouse model of lung fibrosis induced via the infusion of human IPF LFs, because the bleomycin installation model does not fully recapitulate the pathogenesis of IPF. Using the humanized mouse model, we found that intravascular administration of ADSCs also improved fibrotic changes in the lungs. These findings suggest that ADSCs are a promising therapeutic candidate for IPF.

Indexed as

Adipose TissueFibroblastsIdiopathic Pulmonary FibrosisLungMesenchymal Stem CellsAnimalsBleomycinDisease Models, AnimalHumansMaleMAP Kinase Signaling SystemMesenchymal Stem Cell TransplantationMiceMice, Inbred C57BLBleomycinADSCcollagenhumanized miceIPFPTPRR

Identifiers

PMID39768142
PMCPMC11674916

What OpenQuestion holds

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.