Evidence map›Paper›PMID 39702195›Full record

ReviewReproductive biology and endocrinology : RB&E2024

Müllerian anomalies and endometriosis: associations and phenotypic variations.

Surya Bhamidipaty-Pelosi, Isaac Kyei-Barffour, Marianna Volpert, Nora O'Neill, Alyssa Grimshaw, Lars Eriksson, Alla Vash-Margita, Emanuele Pelosi

Abstract readReview
In one paragraph

Review in Reproductive biology and endocrinology : RB&E, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 10 papers.

0numbers the graph read from it
0cells of the map it votes in
10citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

10 citing papers in PubMed.

  1. Article
  2. Article
  3. Article
  4. Article
  5. Review
  6. Article
  7. Article
  8. Article
  9. Article
  10. The Role of Adenogenesis Factors in the Pathogenesis of Endometriosis.International journal of molecular sciences · 2025
    Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors.

Surya Bhamidipaty-Pelosi *Department of Clinical Pharmacology, Indiana University School of Medicine, Indianapolis, IN, USA.
Isaac Kyei-Barffour *Institute for Molecular Bioscience, The University of Queensland, Brisbane, QLD, Australia.
Marianna VolpertSchool of Biomedical Sciences, Queensland University of Technology, Brisbane, QLD, Australia.
Nora O'NeillDivision of Pediatric and Adolescent Gynecology, Department of Obstetrics, Gynecology and Reproductive Sciences, Yale University School of Medicine, New Haven, CT, USA.
Alyssa GrimshawHarvey Cushing/John Hay Whitney Medical Library, Yale University, New Haven, CT, USA.
Lars ErikssonHerston Health Sciences Library, The University of Queensland, Brisbane, QLD, Australia.
Alla Vash-MargitaDivision of Pediatric and Adolescent Gynecology, Department of Obstetrics, Gynecology and Reproductive Sciences, Yale University School of Medicine, New Haven, CT, USA.
Emanuele PelosiCentre for Clinical Research, The University of Queensland, Brisbane, QLD, Australia. epelosi@iu.edu.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Müllerian anomalies are congenital conditions characterized by the incomplete development of the female reproductive tract. Women affected by Müllerian anomalies often display additional malformations of the renal, skeletal, and cardiovascular system, and are at a higher risk for infertility and adverse pregnancy outcomes. Several Müllerian anomalies have been reported in association with endometriosis, but it is unclear if all classes or anatomical variations are associated with the disease. Most importantly, both Müllerian anomalies and endometriosis can manifest with a wide degree of variability, adding further complexity to their poorly defined relationship. Retrograde menstruation occurring in obstructive Müllerian anomalies is a well-accepted mechanism for the development of endometriosis. However, endometriosis can occur following surgical correction of the anomaly or in the absence of obstruction. This suggests that other mechanisms may be involved, although the specific pathogenesis remains elusive. This review provides a comprehensive summary of the current state of clinical research on endometriosis in Müllerian anomalies. This review also highlights research and knowledge gaps, informing the development of future experimental designs to address current limitations including heterogeneity of phenotypes, variable comorbidities, and lack of genetic information.

Indexed as

EndometriosisMullerian DuctsPhenotypeFemaleHumansInfertility, FemalePregnancyUrogenital AbnormalitiesbicornuatedidelphysendometriosisMRKH syndromeMüllerian anomaliesOHVIRAseptateunicornuate

Identifiers

PMID39702195
PMCPMC11656925

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC-ND
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.