Evidence map›Paper›PMID 39665979›Full record

ArticleAnnals of hematology2024

Post-transplant transient abnormal myelopoiesis evolving from a GATA1 mutant clone in umbilical cord blood.

Yusuke Kubota, Masatoshi Sakurai, Yasuhito Nannya, Yasunori Kogure, Kohei Shiroshita, Shinya Fujita, Kentaro Yamaguchi, Kota Mizuno, Jun Kato, Takehiko Mori and 2 more

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Article in Annals of hematology, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

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1 · What the graph read from it

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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

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3 · Its place in the literature

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4 · The record

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5 · Who and what money

Authors and funding

12 authors.

Yusuke KubotaDivision of Hematology, Department of Medicine, Keio University School of Medicine, Tokyo, Japan.
Masatoshi SakuraiDivision of Hematology, Department of Medicine, Keio University School of Medicine, Tokyo, Japan.
Yasuhito NannyaDepartment of Pathology and Tumor Biology, Graduate School of Medicine, Kyoto University, Kyoto, Japan.
Yasunori KogureDivision of Molecular Oncology, National Cancer Center Research Institute, Tokyo, Japan.
Kohei ShiroshitaDivision of Hematology, Department of Medicine, Keio University School of Medicine, Tokyo, Japan.
Shinya FujitaDivision of Hematology, Department of Medicine, Keio University School of Medicine, Tokyo, Japan.
Kentaro YamaguchiDivision of Hematology, Department of Medicine, Keio University School of Medicine, Tokyo, Japan.
Kota MizunoDivision of Hematology, Department of Medicine, Keio University School of Medicine, Tokyo, Japan.
Jun KatoDivision of Hematology, Department of Medicine, Keio University School of Medicine, Tokyo, Japan.
Takehiko MoriDivision of Hematology, Department of Medicine, Keio University School of Medicine, Tokyo, Japan.
Seishi OgawaDepartment of Pathology and Tumor Biology, Graduate School of Medicine, Kyoto University, Kyoto, Japan.
Keisuke KataokaDivision of Hematology, Department of Medicine, Keio University School of Medicine, Tokyo, Japan. kkataoka-tky@umin.ac.jp.

Funding

Japan Agency for Medical Research and Development JP23ek0109617
6 · The paper itself

Abstract

Transient abnormal myelopoiesis (TAM) generally affects newborns with Down syndrome and is associated with constitutional trisomy 21 and a somatic GATA1 mutation. Here we describe a case of TAM which evolved after umbilical cord blood transplantation (UCBT), whose origin was identified as a GATA1 mutation-harboring clone in umbilical cord blood (UCB) by detailed genetic analyses. A 58-year-old male who received UCBT for peripheral T-cell lymphoma presented progressive anemia and thrombocytopenia, and leukocytosis with blast cells in the peripheral blood (PB). Bone marrow (BM) aspiration showed granulocytic and megakaryocytic dysplasia with excess blasts whose karyotype was trisomy 21. Short tandem repeat analysis showed complete donor chimerism. He was initially diagnosed as donor-derived myelodysplastic syndrome (MDS) and treated with azacitidine, followed by secondary transplantation using unrelated BM, providing durable complete remission. Retrospective targeted-capture sequencing analysis of PB/BM samples collected at multiple timepoints identified trisomy 21 and a GATA1 mutation, suggestive of a diagnosis of donor cell-derived TAM (DC-TAM). Importantly, a minor clone with the same GATA1 mutation was detected in UCB by droplet digital PCR. DC-TAM is a rare UCBT-related complication which resembles MDS, but the identification of GATA1 mutation may be useful for its diagnosis. Our genetic analyses revealed that a pre-existing clone in UCB may contribute to the development of donor cell-derived hematologic neoplasms, highlighting the potential relevance of genetic screening of donor UCB.

Indexed as

Cord Blood Stem Cell TransplantationGATA1 Transcription FactorLeukemoid ReactionMutationDown SyndromeFetal BloodHumansMaleMiddle AgedMyelodysplastic SyndromesGATA1 protein, humanGATA1 Transcription FactorDonor cell-derived hematologic neoplasmsDown syndromeDroplet digital PCRTargeted-capture sequencingTransient abnormal myelopoiesisUmbilical cord blood transplantation

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.