ReviewScience progress
HXK, SnRK1, and TOR signaling in plants: Unraveling mechanisms of stress response and secondary metabolism.
Review in Science progress. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 16 papers.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
16 citing papers in PubMed.
- Review
- Integrated Comparative Genomics and Population Genomics Reveal the Genomic and Epigenomic Basis of Barley (Plants (Basel, Switzerland) · 2026Article
- Phosphorylation networks as regulatory hubs in plant stress signaling: kinase dynamics, crosstalk, and network plasticity.Plant cell reports · 2026Review
- Plant Immunometabolism: Metabolic Reprogramming Linking Developmental Signaling and Defense Metabolites.International journal of molecular sciences · 2026Review
- Signaling mechanisms governing the environmental modulation of fruit quality development.Horticulture research · 2026Article
- Genomic insight of plant pathogen, begomovirus and vector cross-talk: an emerging strategy to tackle the leaf curl disease.Molecular biology reports · 2026Review
- Article
- Navigating heavy metal stress: emerging roles of TOR and SnRK signaling in plant tolerance.Frontiers in plant science · 2026Review
- Autophagy in plants: molecular mechanisms and roles in abiotic stress responses.Frontiers in plant science · 2026Review
- Decoding the sugar-strigolactone crosstalk: new frontier in plant growth and stress resilience.Horticulture research · 2026Article
- Decapitation Rapidly Triggers Axillary Bud Release via Regulatory Network Reprogramming inPlants (Basel, Switzerland) · 2025Article
- Calcium Reduces Fruit Abscission inPlants (Basel, Switzerland) · 2025Article
- Arabidopsis Ubiquitin E3 Ligase AtCHYR1 Promotes ROS Production in Plant Responses to Sugar Availability.Plants (Basel, Switzerland) · 2025Article
- Nitrogen-Driven Orchestration of Lateral Root Development: Molecular Mechanisms and Systemic Integration.Biology · 2025Review
- Genomic Analysis of Hexokinase Genes in Foxtail Millet (International journal of molecular sciences · 2025Article
- Article
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
3 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
As sessile photoautotrophs, plants constantly encounter diverse environmental stresses. Recent research has focused on elucidating sugar and energy signaling mediated by hexokinase (HXK), sucrose non-fermenting 1-related protein kinase 1 (SnRK1), and the target of rapamycin (TOR) and assessing its intricate interplay with hormones and secondary metabolism. HXK serves as a pivotal regulator of glucose sensing and metabolism. It affects plant growth and development in response to nutrient availability. SnRK1 acts as a vital energy sensor that regulates metabolic adjustments during stress to bolster plant resilience. Moreover, TOR integrates nutrient signals to finely modulate growth and development, balancing cellular metabolism and resource allocation. Understanding the functions of HXK, SnRK1, and TOR can provide profound insights into plant adaptation mechanisms and open promising avenues for leveraging biotechnological strategies to enhance the stress tolerance and nutritional value of crops. This narrative review focuses on recent advancements in the molecular mechanisms of HXK, SnRK1, and TOR and explores their potential applications in agricultural biotechnology.
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What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.