Article in American journal of respiratory cell and molecular biology, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.
0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
2 · The registry
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
5 · Who and what money
Authors and funding
7 authors.
Alexandra B PincusDepartment of Pediatrics, University of California San Francisco, San Francisco, California; and.ORCID 0000-0002-4373-0543
Aubrey B PierceDivision of Pulmonary, Allergy, and Critical Care, Oregon Health & Science University, Portland, Oregon.
Nicole KappelDivision of Pulmonary, Allergy, and Critical Care, Oregon Health & Science University, Portland, Oregon.
Katie M LeboldDivision of Pulmonary, Allergy, and Critical Care, Oregon Health & Science University, Portland, Oregon.
Matthew G DrakeDivision of Pulmonary, Allergy, and Critical Care, Oregon Health & Science University, Portland, Oregon.ORCID 0000-0001-5476-0361
Allison D FryerDivision of Pulmonary, Allergy, and Critical Care, Oregon Health & Science University, Portland, Oregon.
David B JacobyDivision of Pulmonary, Allergy, and Critical Care, Oregon Health & Science University, Portland, Oregon.
Funding
Identifying mechanisms and reversibility of eosinophil-induced airway hyperinnervation in asthmaR01HL155623 · NHLBI · OREGON HEALTH & SCIENCE UNIVERSITY · PI DRAKE, MATTHEW G. · 2021 to 2025
$3.3M
Prenatal control of offspring airway responsivenessR01HL144008 · NHLBI · OREGON HEALTH & SCIENCE UNIVERSITY · PI JACOBY, DAVID B · 2019 to 2022
$2.4M
Mechanisms of tachykinergic nerve-mediated severe bronchoconstriction and inflammationR01HL171226 · NHLBI · OREGON HEALTH & SCIENCE UNIVERSITY · PI Matthew G. Drake, ALLISON Deborah FRYER · 2024 to 2026
$2.3M
Insulin Modulates Parasympathetic Nerve Control of LungsR01HL131525 · NHLBI · OREGON HEALTH & SCIENCE UNIVERSITY · PI FRYER, ALLISON DEBORAH · 2017 to 2020
$1.9M
An optogenetic approach to airway researchR21AI152498 · NIAID · OREGON HEALTH & SCIENCE UNIVERSITY · PI JACOBY, DAVID B · 2020 to 2021
$424k
Airway sensory nerve changes in a mouse model of maternal allergen exposureF30HL145906 · NHLBI · OREGON HEALTH & SCIENCE UNIVERSITY · PI PINCUS, ALEXANDRA BOWMAN · 2019 to 2022
Airway hyperreactivity in asthma is mediated by airway nerves, including sensory nerves in airway epithelium and parasympathetic nerves innervating airway smooth muscle. Isolating the function of these two nerve populations
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.
Parasympathetic Airway Hyperreactivity Is Enhanced in Acute but Not Chronic Eosinophilic Asthma Mouse Models. · full record | OpenQuestion