Evidence map›Paper›PMID 39620359›Full record

ArticleGut microbes

Defective

Víctor A López-Agudelo, Maren Falk-Paulsen, Richa Bharti, Ateequr Rehman, Felix Sommer, Eike Matthias Wacker, David Ellinghaus, Anne Luzius, Laura Katharina Sievers, Manuel Liebeke and 2 more

Abstract read
In one paragraph

Article in Gut microbes. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 4 papers.

0numbers the graph read from it
0cells of the map it votes in
4citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

4 citing papers in PubMed.

  1. Review
  2. Review
  3. Review
  4. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

12 authors.

Víctor A López-AgudeloInstitute of Clinical Molecular Biology, Christian-Albrechts-University, and University Hospital Schleswig-Holstein, Kiel, Germany.
Maren Falk-PaulsenInstitute of Clinical Molecular Biology, Christian-Albrechts-University, and University Hospital Schleswig-Holstein, Kiel, Germany.
Richa BhartiInstitute of Clinical Molecular Biology, Christian-Albrechts-University, and University Hospital Schleswig-Holstein, Kiel, Germany.
Ateequr RehmanInstitute of Clinical Molecular Biology, Christian-Albrechts-University, and University Hospital Schleswig-Holstein, Kiel, Germany.
Felix SommerInstitute of Clinical Molecular Biology, Christian-Albrechts-University, and University Hospital Schleswig-Holstein, Kiel, Germany.
Eike Matthias WackerInstitute of Clinical Molecular Biology, Christian-Albrechts-University, and University Hospital Schleswig-Holstein, Kiel, Germany.
David EllinghausInstitute of Clinical Molecular Biology, Christian-Albrechts-University, and University Hospital Schleswig-Holstein, Kiel, Germany.
Anne LuziusInstitute of Clinical Molecular Biology, Christian-Albrechts-University, and University Hospital Schleswig-Holstein, Kiel, Germany.
Laura Katharina SieversDepartment of General Internal Medicine, Christian-Albrechts-University and University Hospital Schleswig-Holstein, Kiel, Germany.
Manuel LiebekeDepartment for Metabolomics, Institute for Human Nutrition and Food Science, University of Kiel, Kiel, Germany.
Arthur KaserCambridge Institute of Therapeutic Immunology and Infectious Disease (CITIID), Jeffrey Cheah Biomedical Centre, and Division of Gastroenterology and Hepatology, Department of Medicine, University of Cambridge, Cambridge, UK.
Philip RosenstielInstitute of Clinical Molecular Biology, Christian-Albrechts-University, and University Hospital Schleswig-Holstein, Kiel, Germany.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Throughout gestation, the female body undergoes a series of transformations, including profound alterations in intestinal microbial communities. Changes gradually increase toward the end of pregnancy and comprise reduced α-diversity of microbial communities and an increased propensity for energy harvest. Despite the importance of the intestinal microbiota for the pathophysiology of inflammatory bowel diseases, very little is known about the relationship between these microbiota shifts and pregnancy-associated complications of the disease. Here, we explored the longitudinal dynamics of gut microbiota composition and functional potential during pregnancy and after lactation in

Indexed as

Autophagy-Related ProteinsFecesGastrointestinal MicrobiomeAnimalsBacteriaChemokine CXCL1Crohn DiseaseEpithelial CellsFemaleIntestinal MucosaMetagenomicsMiceMice, Inbred C57BLPregnancyRNA, Ribosomal, 16SAtg16l1 protein, mouseAutophagy-Related ProteinsChemokine CXCL1RNA, Ribosomal, 16SAtg16l1IBDmicrobiotaPregnancy

Identifiers

PMID39620359
PMCPMC11622647

What OpenQuestion holds

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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.