Evidence map›Paper›PMID 39590605›Full record

ReviewJournal of personalized medicine2024

Electrophysiologic Considerations in Adult Patients with Ebstein's Anomaly.

Ingrid Hsiung, Olubadewa A Fatunde, Komandoor Srivathsan, Malini Madhavan, David S Majdalany

Abstract readReview
In one paragraph

Review in Journal of personalized medicine, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors.

Ingrid HsiungDepartment of Cardiovascular Medicine, University of Southern California, Los Angeles, CA 90007, USA.ORCID 0000-0002-2347-7261
Olubadewa A FatundeDepartment of Cardiovascular Medicine, Mayo Clinic, Scottsdale, AZ 85259, USA.ORCID 0000-0002-2869-5022
Komandoor SrivathsanDepartment of Cardiovascular Medicine, Mayo Clinic, Scottsdale, AZ 85259, USA.
Malini MadhavanDepartment of Cardiovascular Medicine, Mayo Clinic, Rochester, MN 55905, USA.
David S MajdalanyDepartment of Cardiovascular Medicine, Mayo Clinic, Rochester, MN 55905, USA.ORCID 0000-0002-5472-6689

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Ebstein's anomaly (EA) is a rare cyanotic form of congenital heart disease (CHD) characterized by apical displacement of the tricuspid valve, with resultant hemodynamic and electrical manifestations. The severity of symptoms is determined by the degree of apical displacement and deformity and incompetence of the tricuspid valve. As a result, patients with EA can be severely symptomatic during infancy and childhood or can be incidentally discovered in the sixth or seventh decade of life. Hallmarks of Ebstein's anomaly include progressive cyanosis, right-sided heart failure, and tachyarrhythmias, among which tachyarrhythmias (most commonly atrial, but also ventricular) are the most common presenting symptoms in Ebstein's anomaly patients during adulthood. This review aims to provide insight into the genetic and electrophysiological (EP) basis underlying the tachyarrhythmias encountered when managing patients with EA.

Indexed as

anomalycongenital heart diseasecyanotic heart diseaseelectrophysiology

Identifiers

PMID39590605
PMCPMC11596009

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.