Evidence map›Paper›PMID 39527185›Full record

ArticleJournal of microbiology (Seoul, Korea)2024

H-NS is a Transcriptional Repressor of the CRISPR-Cas System in Acinetobacter baumannii ATCC 19606.

Kyeongmin Kim, Md Maidul Islam, Seunghyeok Bang, Jeongah Kim, Chung-Young Lee, Je Chul Lee, Minsang Shin

Abstract read
PubMed Publisher
In one paragraph

Article in Journal of microbiology (Seoul, Korea), 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.

0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

2 citing papers in PubMed.

  1. Article
  2. The H-NS homologues MvaT and MvaU repress CRISPR-Cas inPhilosophical transactions of the Royal Society of London. Series B, Biological sciences · 2025
    Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors.

Kyeongmin Kim *Department of Microbiology, School of Medicine, Kyungpook National University, 680 Gukchaebosang- Ro, Jung-gu, Daegu, 41944, Republic of Korea.
Md Maidul Islam *Department of Microbiology, School of Medicine, Kyungpook National University, 680 Gukchaebosang- Ro, Jung-gu, Daegu, 41944, Republic of Korea.
Seunghyeok Bang *Department of Microbiology, School of Medicine, Kyungpook National University, 680 Gukchaebosang- Ro, Jung-gu, Daegu, 41944, Republic of Korea.
Jeongah KimDepartment of Microbiology, School of Medicine, Kyungpook National University, 680 Gukchaebosang- Ro, Jung-gu, Daegu, 41944, Republic of Korea.
Chung-Young LeeDepartment of Microbiology, School of Medicine, Kyungpook National University, 680 Gukchaebosang- Ro, Jung-gu, Daegu, 41944, Republic of Korea.
Je Chul LeeDepartment of Microbiology, School of Medicine, Kyungpook National University, 680 Gukchaebosang- Ro, Jung-gu, Daegu, 41944, Republic of Korea.
Minsang ShinDepartment of Microbiology, School of Medicine, Kyungpook National University, 680 Gukchaebosang- Ro, Jung-gu, Daegu, 41944, Republic of Korea. shinms@knu.ac.kr.ORCID http://orcid.org/0000-0001-8679-446X

Funding

Ministry of Science and ICT, South Korea 2022R1A2C2010683
6 · The paper itself

Abstract

Acinetobacter baumannii is a multidrug-resistant opportunistic pathogen primarily associated with hospital-acquired infections. The bacterium can gain multidrug resistance through several mechanisms, including horizontal gene transfer. A CRISPR-Cas system including several Cas genes could restrict the horizontal gene transfer. However, the molecular mechanism of CRISPR- Cas transcriptional regulation remains unclear. We identified a type I-F CRISPR-Cas system in A. baumannii ATCC 19606

Indexed as

Acinetobacter baumanniiBacterial ProteinsCRISPR-Cas SystemsGene Expression Regulation, BacterialPromoter Regions, GeneticCRISPR-Associated ProteinsDNA-Binding ProteinsRepressor ProteinsBacterial ProteinsCRISPR-Associated ProteinsDNA-Binding ProteinsH-NS protein, bacteriaRepressor ProteinsAcinetobacter baumanniiCas3CRISPR-CasH-NSTranscriptional Regulation

Identifiers

PMID39527185

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.