Evidence map›Paper›PMID 39464593›Full record

ArticlePostepy w kardiologii interwencyjnej = Advances in interventional cardiology2024

Cardiac troponin I release after transcatheter closure of atrial septal defects is associated with supraventricular arrhythmias on early follow-up.

Paweł Prochownik, Klaudia Bielecka, Tadeusz Przewłocki, Zuzanna Sachajko, Urszula Gancarczyk, Piotr Wilkołek, Michał Tworek, Piotr Podolec, Larysa Bielecka, Monika Komar

Abstract read
In one paragraph

Article in Postepy w kardiologii interwencyjnej = Advances in interventional cardiology, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed.

  1. From bench to bedside and back: translational cardiovascular interventions in veterinary medicine.Postepy w kardiologii interwencyjnej = Advances in interventional cardiology · 2025
    Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

10 authors.

Paweł ProchownikDepartment of Cardiac and Vascular Diseases, John Paul II Hospital, Institute of Cardiology, Krakow, Poland.
Klaudia BieleckaDepartment of Cardiac and Vascular Diseases, John Paul II Hospital, Institute of Cardiology, Krakow, Poland.
Tadeusz PrzewłockiDepartment of Cardiac and Vascular Diseases, John Paul II Hospital, Institute of Cardiology, Krakow, Poland.
Zuzanna SachajkoFaculty of Medicine, Jagiellonian University Medical College, Krakow, Poland.
Urszula GancarczykDepartment of Cardiac and Vascular Diseases, John Paul II Hospital, Institute of Cardiology, Krakow, Poland.
Piotr WilkołekDepartment of Cardiac and Vascular Diseases, John Paul II Hospital, Institute of Cardiology, Krakow, Poland.
Michał TworekDepartment of Cardiac and Vascular Diseases, John Paul II Hospital, Institute of Cardiology, Krakow, Poland.
Piotr PodolecDepartment of Cardiac and Vascular Diseases, John Paul II Hospital, Institute of Cardiology, Krakow, Poland.
Larysa BieleckaFaculty of Medicine, Jagiellonian University Medical College, Krakow, Poland.
Monika KomarDepartment of Cardiac and Vascular Diseases, John Paul II Hospital, Institute of Cardiology, Krakow, Poland.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Introduction: Atrial septal defects (ASD) are prevalent congenital heart anomalies found in the adult population. Percutaneous ASD closure has become a routine clinical practice. Elevation of postprocedural transient cardiac biomarkers and exacerbation of supraventricular arrhythmias have been reported in the subject literature. Aim: To explore the relationship between cardiac troponin I (cTnI) elevation and supraventricular ectopy (SVE) following percutaneous closure of secundum atrial septal defect (ASD) in adult patients. Material and methods: 600 consecutive patients who underwent successful transcatheter ASD secundum closure were analyzed. Serum levels of cTnI were measured before and within 72 h of device implantation. 24-hour Holter monitoring was performed before the procedure, at 1 month, and at 6 months of follow-up. Results: SVE burden increased 1 month after the procedure (median 1021.00; min.-max. 11.00-29 862.00) compared to baseline values (median 146.00; min.-max. 0-1865.00; Conclusions: A significant increase of cTnI is noted frequently after transcatheter ASD closure and seems to predict exacerbation in SVE burden on short-term follow up. The independent risk factors of cTnI rise are prolonged procedure duration and larger device sizes.

Indexed as

arrhythmiacongenital heart diseaseechocardiographyelectrocardiographyinterventional cardiologymyocardial damage

Identifiers

PMID39464593
PMCPMC11506394

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC-SA
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.