Evidence map›Paper›PMID 39453744›Full record

ArticleProceedings of the National Academy of Sciences of the United States of America2024

Intrinsically disordered region amplifies membrane remodeling to augment selective ER-phagy.

Sergio Alejandro Poveda-Cuevas, Kateryna Lohachova, Borna Markusic, Ivan Dikic, Gerhard Hummer, Ramachandra M Bhaskara

Abstract read
In one paragraph

Article in Proceedings of the National Academy of Sciences of the United States of America, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 12 papers.

0numbers the graph read from it
0cells of the map it votes in
12citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

12 citing papers in PubMed.

  1. Emerging mechanisms of ATG8-independent non-canonical autophagy.Journal of physiology and biochemistry · 2026
    Review
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  12. Intrinsically disordered region amplifies membrane remodeling to augment selective ER-phagy.Proceedings of the National Academy of Sciences of the United States of America · 2024
    Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors.

Sergio Alejandro Poveda-CuevasGoethe University Frankfurt, School of Medicine, Institute of Biochemistry II, Frankfurt am Main 60590, Germany.ORCID 0000-0002-3087-6674
Kateryna LohachovaGoethe University Frankfurt, School of Medicine, Institute of Biochemistry II, Frankfurt am Main 60590, Germany.ORCID 0000-0001-7826-8320
Borna MarkusicGoethe University Frankfurt, School of Medicine, Institute of Biochemistry II, Frankfurt am Main 60590, Germany.ORCID 0009-0007-8302-812X
Ivan DikicGoethe University Frankfurt, School of Medicine, Institute of Biochemistry II, Frankfurt am Main 60590, Germany.ORCID 0000-0001-8156-9511
Gerhard HummerMax-Planck Institute of Biophysics, Department of Theoretical Biophysics, Frankfurt am Main 60438, Germany.ORCID 0000-0001-7768-746X
Ramachandra M BhaskaraGoethe University Frankfurt, School of Medicine, Institute of Biochemistry II, Frankfurt am Main 60590, Germany.ORCID 0000-0002-7742-0391

Funding

Deutsche Forschungsgemeinschaft (DFG) 259130777-SFB1177ERC ER-REMODELHessian Ministry of Science and Arts EnABLE
6 · The paper itself

Abstract

Intrinsically disordered regions (IDRs) play a pivotal role in organellar remodeling. They transduce signals across membranes, scaffold signaling complexes, and mediate vesicular traffic. Their functions are regulated by constraining conformational ensembles through specific intra- and intermolecular interactions, physical tethering, and posttranslational modifications. The endoplasmic reticulum (ER)-phagy receptor FAM134B/RETREG1, known for its reticulon homology domain (RHD), includes a substantial C-terminal IDR housing the LC3 interacting motif. Beyond engaging the autophagic machinery, the function of the FAM134B-IDR is unclear. Here, we investigate the characteristics of the FAM134B-IDR by extensive modeling and molecular dynamics simulations. We present detailed structural models for the IDR, mapping its conformational landscape in solution and membrane-anchored configurations. Our analysis reveals that depending on the membrane anchor, the IDRs collapse onto the membrane and induce positive membrane curvature to varying degrees. The charge patterns underlying this Janus-like behavior are conserved across other ER-phagy receptors. We found that IDRs alone are sufficient to sense curvature. When combined with RHDs, they intensify membrane remodeling and drive efficient protein clustering, leading to faster budding, thereby amplifying RHD remodeling functions. Our simulations provide a perspective on IDRs of FAM134B, their Janus-like membrane interactions, and the resulting modulatory functions during large-scale ER remodeling.

Indexed as

Endoplasmic ReticulumIntrinsically Disordered ProteinsMembrane ProteinsMolecular Dynamics SimulationAutophagyCell MembraneHumansIntracellular Signaling Peptides and ProteinsProtein DomainsIntracellular Signaling Peptides and ProteinsIntrinsically Disordered ProteinsMembrane ProteinsRETREG1 protein, humanconformational entropycurvature inductionER remodelingIDRsstructural ensemble

Identifiers

PMID39453744
PMCPMC11536123

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC-ND
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.