ReviewCommunications biology2024
The unique role of anosognosia in the clinical progression of Alzheimer's disease: a disorder-network perspective.
Review in Communications biology, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 4 papers.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Who cites it
4 citing papers in PubMed.
- Error unawareness as a driver of cognitive decline in individuals at risk for Alzheimer's disease.Communications biology · 2026Article
- Denial of Hardware-Related Skin Erosion in Patients with Effective Deep Brain Stimulation: A Novel Phenomenon.Stereotactic and functional neurosurgery · 2026Article
- Deficits in reaching movements under visual interference as a novel diagnostic marker for mild cognitive impairment.Scientific reports · 2025Article
- Neuropsychological correlates of reduced self-awareness of functional competency in persons with subjective memory complaints, mild cognitive impairment, and early probable Alzheimer's dementia.Frontiers in neurology · 2025Article
Corrections and comments
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Authors and funding
2 authors.
Funding
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Abstract
Alzheimer's disease (AD) encompasses a long continuum from a preclinical phase, characterized by neuropathological alterations albeit normal cognition, to a symptomatic phase, marked by its clinical manifestations. Yet, the neural mechanisms responsible for cognitive decline in AD patients remain poorly understood. Here, we posit that anosognosia, emerging from an error-monitoring failure due to early amyloid-β deposits in the posterior cingulate cortex, plays a causal role in the clinical progression of AD by preventing patients from being aware of their deficits and implementing strategies to cope with their difficulties, thus fostering a vicious circle of cognitive decline.
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