Assessment of the contribution of

Pratibha Banerjee, Harinder Singh, Priyanka Tiwari, Ajit Sood, Vandana Midha, Gursewak Singh, B K Thelma, Sabyasachi Senapati

Abstract read

PubMed

In one paragraph

Article in Journal of genetics, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it

0cells of the map it votes in

1citing papers in PubMed

–field-weighted citation impact

1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed.

Identification and Association ofCurrent issues in molecular biology · 2025
Article

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors.

Pratibha BanerjeeImmunogenomics Laboratory, Department of Human Genetics and Molecular Medicine, Central University of Punjab, Bathinda 151 401, India. sabyasachi1012@gmail.com.

Harinder Singh

Priyanka Tiwari

Ajit Sood

Vandana Midha

Gursewak Singh

B K Thelma

Sabyasachi Senapati

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Vitamin-D deficiency (VDD) is a global health concern. It is known to play a critical role in the immunomodulation, and thus, its metabolism could be investigated to unravel its contribution in common immune-mediated diseases, e.g., celiac disease (CD). Genotyping of SNPs from vitamin D receptor (

Indexed as

AllelesCeliac DiseaseGenetic Predisposition to DiseasePolymorphism, Single NucleotideReceptors, CalcitriolVitamin D-Binding ProteinAdolescentAdultCase-Control StudiesChildFemaleGene FrequencyGenotypeHaplotypesHumansMaleGC protein, humanReceptors, CalcitriolVDR protein, humanVitamin DVitamin D-Binding Protein

Identifiers

PMID39445578

What OpenQuestion holds

Textmetadata

Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.