ArticleAnimal models and experimental medicine2024
Histopathologic observations in a coccidiosis model of Eimeria tenella.
Article in Animal models and experimental medicine, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 9 papers.
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9 citing papers in PubMed.
- Poultry Coccidiosis in and Around Arba Minch, Southern Ethiopia: A Study Based on Coprological and Postmortem Findings.Immunity, inflammation and disease · 2026Article
- Dietary Artemisia annua leaf powder modulates intestinal response and cecal microbiota of broiler chickens during peak Eimeria infection.Poultry science · 2026Article
- Pathogenic mechanism of Eimeria tenella EtMIC2 promotes Eimeria tenella invasion and inhibits host cell apoptosis through binding to the ITGAV receptor.Poultry science · 2026Article
- Anticoccidial Resistance inVeterinary medicine international · 2026Article
- An Integrated Model of Biphasic Apoptosis in Avian Coccidiosis: Molecular Networks and Host-Parasite Interplay.Animals : an open access journal from MDPI · 2025Review
- Prevalence and anticoccidial drug sensitivity ofVeterinary world · 2025Article
- Evaluation of the Effectiveness of a Phytogenic Supplement (Alkaloids and Flavonoids) in the Control ofAnimals : an open access journal from MDPI · 2025Article
- Knockdown of annexin A2 enhances the host cell apoptosis induced byFrontiers in veterinary science · 2025Article
- Histopathologic observations in a coccidiosis model of Eimeria tenella.Animal models and experimental medicine · 2024Article
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17 authors.
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Abstract
backgroundSpecies of the genus Eimeria cause coccidiosis in chickens, resulting in a huge burden to the poultry industry worldwide. Eimeria tenella is one of the most prevalent chicken coccidia in China, and E. tenella infection causes hemorrhagic cecitis.
methodsUsing an established model of coccidiosis in chickens combined with necropsy, imaging of pathological tissue sections, and other techniques, we evaluated the gross and microscopic lesions of cecal tissue within 15 days after inoculation with sporulated oocysts and described the endogenetic developmental process and relationship between E. tenella infection and enteritis development in chickens.
resultsWe observed three generations of merogony and gamogony in E. tenella. We observed gross lesions in the cecum from 84 hpi (hours post inoculation) and microscopic lesions from 60 hpi. The lesions in the cecum mainly exhibited hemorrhagic enteritis. Their severity increased with the onset of the second generation of merogony. The lesions began to alleviate by the end of the endogenous stages of E. tenella.
conclusionWe show, for the first time, the complete observation of a series of changes in enteritis caused by 5 × 10
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