Evidence map›Paper›PMID 39424418›Full record

ArticleJournal of the American Heart Association2024

Noise Exposure in the Workplace, Genetic Susceptibility, and Incidence of Atrial Fibrillation: A Prospective Cohort Study.

Wenzhen Li, Junjie Zhu, Xingjie Hao, Zhaomin Chen, Dongming Wang

Abstract read
In one paragraph

Article in Journal of the American Heart Association, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.

0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

2 citing papers in PubMed.

  1. Article
  2. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors.

Wenzhen LiDepartment of Occupational & Environmental Health, School of Public Health Tongji Medical College, Huazhong University of Science and Technology Wuhan Hubei China.
Junjie ZhuDepartment of Epidemiology and Health Statistics, School of Public Health Dali University Dali Yunnan China.ORCID 0000-0002-7662-4915
Xingjie HaoDepartment of Epidemiology and Biostatistics, School of Public Health Tongji Medical College, Huazhong University of Science and Technology Wuhan Hubei China.ORCID 0000-0003-1535-9860
Zhaomin ChenDepartment of Occupational & Environmental Health, School of Public Health Tongji Medical College, Huazhong University of Science and Technology Wuhan Hubei China.
Dongming WangDepartment of Occupational & Environmental Health, School of Public Health Tongji Medical College, Huazhong University of Science and Technology Wuhan Hubei China.ORCID 0000-0002-9297-8369

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

backgroundNo study explored the association of noise exposure in the workplace and genetic susceptibility with incidence of atrial fibrillation (AF). We aimed to assess the separate and joint relationship of noise exposure in the workplace and genetic susceptibility with the risk of AF. METHODS AND

resultsWe included 167 577 participants without AF at baseline in UK Biobank. Cox proportional hazards models were used to assess the separate and joint association of noise exposure in the workplace and genetic susceptibility with the risk of AF. During a median follow-up of 11.83 years, we observed 9355 AF cases. Compared with no noise exposure in the workplace, the hazard ratios (HRs) and were 1.08 (95% CI, 0.99-1.18) for noise exposure in the workplace of <1 year, 1.03 (95% CI, 0.95-1.12) for noise exposure in the workplace of around 1 to 5 years, and 1.08 (95% CI, 1.02-1.14) for noise exposure in the workplace of >5 years, respectively, after adjusting for potential confounders. Genetic risk was positively associated with AF, compared with low genetic risk (tertile 1), the HRs were 1.50 (95% CI, 1.41-1.59) for medium genetic risk (tertile 2) and 2.51 (95% CI, 2.38-2.65) for high genetic risk (tertile 3). However, no interaction between noise exposure in the workplace and genetic susceptibility was observed (

conclusionsLong-term noise exposure in the workplace is positively associated with a higher incidence of AF regardless of genetic background.

Indexed as

Atrial FibrillationGenetic Predisposition to DiseaseNoise, OccupationalAdultAgedFemaleGene-Environment InteractionHumansIncidenceMaleMiddle AgedOccupational ExposureProspective StudiesRisk AssessmentRisk FactorsUnited Kingdomatrial fibrillationgenetic susceptibilitynoise exposureworkplace

Identifiers

PMID39424418
PMCPMC11935669

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.