Evidence map›Paper›PMID 39422868›Full record

ReviewBrazilian journal of microbiology : [publication of the Brazilian Society for Microbiology]2024

The mechanisms of Zika virus-induced neuropathogenesis.

Nahla Ahmed Hassaan, Li Xing

Abstract readReview
PubMed Full text at PMC Publisher
In one paragraph

Review in Brazilian journal of microbiology : [publication of the Brazilian Society for Microbiology], 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors.

Nahla Ahmed HassaanInstitutes of Biomedical Sciences, Shanxi University, 92 Wucheng Road, Taiyuan, Shanxi province, 030006, China.
Li XingInstitutes of Biomedical Sciences, Shanxi University, 92 Wucheng Road, Taiyuan, Shanxi province, 030006, China. xingli107@outlook.com.ORCID http://orcid.org/0000-0002-1405-3459

Funding

The four batches innovation project of invigorating medical through science and technology of Shanxi province 2023XM015The Programme of Introducing Talents of Discipline to Universities D21004
6 · The paper itself

Abstract

Zika virus (ZIKV), a flavivirus, is one of the most serious re-emerging pathogens. Growing outbreaks in the Americas have linked ZIKV to significant clinical symptoms including Guillain-Barré syndrome in adults and congenital anomalies in newborns. ZIKV affects brain cells in a variety of ways, mostly apoptosis and cell cycle delays. Modulation of the host's immune reaction and the inflammatory process has also been shown to be involved in ZIKV-induced neurological disorders. This review summarized and discussed the latest advances in ZIKV research to shed fresh light on the multiple mechanisms incolved in ZIKV-induced neuropathogenesis.

Indexed as

Zika VirusZika Virus InfectionAnimalsApoptosisBrainGuillain-Barre SyndromeHumansImmune responseMicrocephalyMitochondrial destructionNeuroinflammationPathogenicityZika virus (ZIKV)

Identifiers

PMID39422868
PMCPMC11711583

What OpenQuestion holds

Textmetadata
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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.