Evidence map›Paper›PMID 39404369›Full record

ArticleCells2024

Dysfunction of Telomeric Cdc13-Stn1-Ten1 Simultaneously Activates DNA Damage and Spindle Checkpoints.

Nathalie Grandin, Michel Charbonneau

Abstract read
PubMed Full text at PMC Publisher
In one paragraph

Article in Cells, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed.

  1. TheCells · 2025
    Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors.

Nathalie GrandinGReD Institute, CNRS UMR6293, INSERM U1103, Faculty of Medicine, University Clermont-Auvergne, 28 Place Henri Dunant, BP 38, 63001 Clermont-Ferrand Cedex, France.
Michel CharbonneauGReD Institute, CNRS UMR6293, INSERM U1103, Faculty of Medicine, University Clermont-Auvergne, 28 Place Henri Dunant, BP 38, 63001 Clermont-Ferrand Cedex, France.ORCID 0000-0002-6684-6151

Funding

LIGUE Nationale contre le Cancer CCAURA
6 · The paper itself

Abstract

Telomeres, the ends of eukaryotic linear chromosomes, are composed of repeated DNA sequences and specialized proteins, with the conserved telomeric Cdc13/CTC1-Stn1-Ten1 (CST) complex providing chromosome stability via telomere end protection and the regulation of telomerase accessibility. In this study,

Indexed as

Cell Cycle ProteinsDNA DamageSaccharomyces cerevisiaeSaccharomyces cerevisiae ProteinsSpindle ApparatusTelomereTelomere-Binding ProteinsChromosomal Proteins, Non-HistoneMutationCdc13 protein, S cerevisiaeCell Cycle ProteinsChromosomal Proteins, Non-HistoneSaccharomyces cerevisiae ProteinsStn1 protein, S cerevisiaeTelomere-Binding ProteinsTEN1 protein, S cerevisiaeBub2 and Mad2 spindle checkpointsbudding yeast telomeresCdc13-Stn1-Ten1 complexcell cycleMec1 DNA damage checkpointSiz1 SUMO E3 ligase

Identifiers

PMID39404369
PMCPMC11475793

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.