Evidence map›Paper›PMID 39368965›Full record

ArticleTranslational psychiatry2024

Basolateral amygdala parvalbumin and cholecystokinin-expressing GABAergic neurons modulate depressive and anxiety-like behaviors.

Muhammad Asim, Huajie Wang, Abdul Waris, Jufang He

Abstract read
In one paragraph

Article in Translational psychiatry, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 17 papers.

0numbers the graph read from it
0cells of the map it votes in
17citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

17 citing papers in PubMed.

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  4. mGluR5 in ECAdvanced science (Weinheim, Baden-Wurttemberg, Germany) · 2026
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors.

Muhammad AsimDepartment of Neuroscience, City University of Hong Kong, Kowloon Tong, Hong Kong. muhamasim5@cityu.edu.hk.ORCID 0000-0002-6912-4461
Huajie WangDepartment of Neuroscience, City University of Hong Kong, Kowloon Tong, Hong Kong.
Abdul WarisDepartment of Biomedical Science, City University of Hong Kong, Kowloon Tong, Hong Kong.
Jufang HeDepartment of Neuroscience, City University of Hong Kong, Kowloon Tong, Hong Kong. jufanghe@cityu.edu.hk.ORCID 0000-0002-4288-5957

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

The basolateral amygdala (BLA) is increasingly recognized as a key regulator of depression and anxiety-like behaviors. However, the specific contribution of individual BLA neurons to these behaviors remains poorly understood. Building on our previous study, which demonstrated increased activity in glutamatergic BLA neurons in response to aversive stimuli and that enhancing inhibition in the BLA can alleviate depressive-like behaviors, we investigated the role of individual BLA GABAergic neurons (BLA

Indexed as

AnxietyBasolateral Nuclear ComplexBehavior, AnimalCholecystokininDepressionGABAergic NeuronsParvalbuminsAnimalsDisease Models, AnimalHindlimb SuspensionMaleMiceMice, Inbred C57BLOptogeneticsSocial DefeatSocial InteractionCholecystokininParvalbuminsSomatostatin

Identifiers

PMID39368965
PMCPMC11455908

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC-ND
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.