Evidence map›Paper›PMID 39363102›Full record

ArticleCommunications biology2024

Sexual dimorphism in a mouse model of Friedreich's ataxia with severe cardiomyopathy.

Lili Salinas, Claire B Montgomery, Francisco Figueroa, Phung N Thai, Nipavan Chiamvimonvat, Gino Cortopassi, Elena N Dedkova

Abstract read
PubMed Full text at PMC Publisher
In one paragraph

Article in Communications biology, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 6 papers.

0numbers the graph read from it
0cells of the map it votes in
6citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

6 citing papers in PubMed.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors.

Lili SalinasDepartment of Molecular Biosciences, University of California, Davis, CA, USA.ORCID 0009-0003-4743-2134
Claire B MontgomeryDepartment of Molecular Biosciences, University of California, Davis, CA, USA.ORCID 0000-0002-1107-1029
Francisco FigueroaDepartment of Molecular Biosciences, University of California, Davis, CA, USA.
Phung N ThaiDepartment of Internal Medicine, University of California, Davis, CA, USA.ORCID 0000-0001-7344-150X
Nipavan ChiamvimonvatDepartment of Internal Medicine, University of California, Davis, CA, USA.
Gino CortopassiDepartment of Molecular Biosciences, University of California, Davis, CA, USA.ORCID 0000-0003-2563-4133
Elena N DedkovaDepartment of Molecular Biosciences, University of California, Davis, CA, USA. ededkova@ucdavis.edu.ORCID 0000-0002-0601-9816

Funding

Functional Interactions of Cardiac Ion ChannelsR01HL085844 · NHLBI · UNIVERSITY OF CALIFORNIA AT DAVIS · PI CHIAMVIMONVAT, NIPAVAN · 2007 to 2025
$7.3M
Training Program In Basic & Translational Cardiovascular ScienceT32HL086350 · NHLBI · UNIVERSITY OF CALIFORNIA AT DAVIS · PI Martin Cadeiras, David A. Liem · 2008 to 2026
$7.1M
Translational Study of Cardiac DysfunctionR01HL085727 · NHLBI · UNIVERSITY OF CALIFORNIA AT DAVIS · PI CHIAMVIMONVAT, NIPAVAN, SIRISH, PADMINI · 2008 to 2022
$5.7M
MItochondrial Dysfunction in Cardiac Hypertrophy and FailureR01HL101235 · NHLBI · JOHNS HOPKINS UNIVERSITY · PI BERS, DONALD M, BLATTER, LOTHAR A · 2010 to 2013
$2.8M
Decoding the enigma of cardiac amplificationR01HL137228 · NHLBI · UNIVERSITY OF CALIFORNIA AT DAVIS · PI CHIAMVIMONVAT, NIPAVAN · 2017 to 2020
$2.2M
Fumarate drugs rescue cardiac dysfunction in mouse models of Friedreich's ataxiaR01HL155907 · NHLBI · UNIVERSITY OF CALIFORNIA AT DAVIS · PI DEDKOVA, ELENA N. · 2021 to 2024
$1.8M
In Vivo Ultrasound Imaging System (VEVO 2100, VisualSonics)S10OD010389 · OD · UNIVERSITY OF CALIFORNIA AT DAVIS · PI CHIAMVIMONVAT, NIPAVAN · 2013 to 2013
$397k
Novel Mechanisms of Cardiac pH Regulation During Ischemia/ReperfusionF32HL149288 · NHLBI · UNIVERSITY OF CALIFORNIA AT DAVIS · PI THAI, PHUNG NHAT · 2019 to 2020
$91k
Metabolomics study in patients post myocardial infarctionI01CX001490 · VA · VA NORTHERN CALIFORNIA HEALTH CARE SYS · PI CHIAMVIMONVAT, NIPAVAN · 2017 to 2021
–
Regulation of ion channels in the heartI01BX000576 · VA · VA NORTHERN CALIFORNIA HEALTH CARE SYS · PI CHIAMVIMONVAT, NIPAVAN · 2009 to 2019
–
BLRD VA I01 BX000576CSRD VA I01 CX001490NHLBI NIH HHS F32 HL149288NHLBI NIH HHS R01 HL085727NHLBI NIH HHS R01 HL085844NHLBI NIH HHS R01 HL137228NHLBI NIH HHS R01 HL155907NHLBI NIH HHS T32 HL086350U.S. Department of Health & Human Services | NIH | National Heart, Lung, and Blood Institute (NHLBI) 1R01HL155907-1
6 · The paper itself

Abstract

Friedreich's ataxia (FA) is an autosomal recessive disorder caused by reduced frataxin (FXN) expression in mitochondria, where the lethal component is cardiomyopathy. Using the conditional Fxn

Indexed as

CardiomyopathiesDisease Models, AnimalFrataxinFriedreich AtaxiaIron-Binding ProteinsMice, KnockoutSex CharacteristicsAnimalsFemaleMaleMiceReceptors, GABATestosteroneBzrp protein, mouseFrataxinIron-Binding ProteinsReceptors, GABATestosterone

Identifiers

PMID39363102
PMCPMC11449905

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.