Evidence map›Paper›PMID 39336763›Full record

ArticleGenes2024

Epidemiological Study on the Interaction between the

Satoshi Sato, Chikara Iino, Takafumi Sasada, Go Soma, Keisuke Furusawa, Kenta Yoshida, Kaori Sawada, Tatsuya Mikami, Shigeyuki Nakaji, Hirotake Sakuraba and 1 more

Abstract read
In one paragraph

Article in Genes, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 4 papers.

0numbers the graph read from it
0cells of the map it votes in
4citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

4 citing papers in PubMed.

  1. Article
  2. Article
  3. Review
  4. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

11 authors.

Satoshi SatoDepartment of Gastroenterology, Hematology, and Clinical Immunology, Hirosaki University Graduate School of Medicine, Hirosaki 036-8562, Japan.ORCID 0000-0001-5569-7093
Chikara IinoDepartment of Gastroenterology, Hematology, and Clinical Immunology, Hirosaki University Graduate School of Medicine, Hirosaki 036-8562, Japan.
Takafumi SasadaDepartment of Gastroenterology, Hematology, and Clinical Immunology, Hirosaki University Graduate School of Medicine, Hirosaki 036-8562, Japan.
Go SomaDepartment of Gastroenterology, Hematology, and Clinical Immunology, Hirosaki University Graduate School of Medicine, Hirosaki 036-8562, Japan.
Keisuke FurusawaDepartment of Gastroenterology, Hematology, and Clinical Immunology, Hirosaki University Graduate School of Medicine, Hirosaki 036-8562, Japan.
Kenta YoshidaDepartment of Gastroenterology, Hematology, and Clinical Immunology, Hirosaki University Graduate School of Medicine, Hirosaki 036-8562, Japan.ORCID 0000-0002-3897-5546
Kaori SawadaDepartment of Preemptive Medicine, Hirosaki University Graduate School of Medicine, Hirosaki 036-8562, Japan.
Tatsuya MikamiDepartment of Preemptive Medicine, Hirosaki University Graduate School of Medicine, Hirosaki 036-8562, Japan.ORCID 0000-0002-6657-6446
Shigeyuki NakajiDepartment of Preemptive Medicine, Hirosaki University Graduate School of Medicine, Hirosaki 036-8562, Japan.ORCID 0000-0002-4190-5151
Hirotake SakurabaDepartment of Gastroenterology, Hematology, and Clinical Immunology, Hirosaki University Graduate School of Medicine, Hirosaki 036-8562, Japan.
Shinsaku FukudaDepartment of Gastroenterology, Hematology, and Clinical Immunology, Hirosaki University Graduate School of Medicine, Hirosaki 036-8562, Japan.

Funding

Japan Science and Technology Agency JPMJCE1302, JPMJCA2201, JPMJF2210JSPS KAKENHI 22K17386
6 · The paper itself

Abstract

Many factors are associated with the development and progression of metabolic dysfunction-associated steatotic liver disease (MASLD); however, genetics and gut microbiota are representative factors. Recent studies have highlighted the link between host genes and the gut microbiota. Although there have been many studies on the separate effects of single nucleotide polymorphisms (SNPs) and gut bacteria on MASLD, few epidemiological studies have examined how SNPs and gut bacteria interact in the development and progression of MASLD. This study aimed to investigate the association between

Indexed as

Gastrointestinal MicrobiomeLipaseMembrane ProteinsPolymorphism, Single NucleotideAcyltransferasesAdultAgedCross-Sectional StudiesFatty LiverFemaleGenotypeHumansMaleMiddle AgedPhospholipases A2, Calcium-IndependentAcyltransferasesLipaseMembrane ProteinsPhospholipases A2, Calcium-IndependentPNPLA3 protein, humangut microbiotaMASLDPNPLA3SNPs

Identifiers

PMID39336763
PMCPMC11430940

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.