ArticleHeliyon2024
Numerical analysis of the effect of Syringomyelia on cerebrospinal fluid dynamics.
Article in Heliyon, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
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Who cites it
2 citing papers in PubMed.
- MRI-derived three-dimensional modelling reveals cervicothoracic subarachnoid space narrowing in syringomyelia-affected cavalier king charles spaniels.BMC veterinary research · 2026Article
- Prediction of time averaged wall shear stress distribution in coronary arteries' bifurcation varying in morphological features via deep learning.Frontiers in physiology · 2025Article
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
2 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Spinal cord enlargement (SCE) includes conditions such as Syringomyelia, tumors, and tumor-like cases of demyelination, edema, or inflammation. These conditions involve fluid-filled cysts, known as syrinx, or masses of tissue, referred to as tumors, which cause increased pressure within the spinal cord (SC) and obstruct cerebrospinal fluid (CSF) circulation. To assess the impact of SCE location and diameter, we constructed fifteen computational SC models, each featuring a SCE placed in one of five probable locations with 20 %, 40 %, and 60 % stenosis. Our objective was to investigate how the location, diameter, and length of the SCE influence CSF velocity pattern and to identify the most critical location in the SC associated with this condition. The results indicated a velocity increase of 0.5 cm/(s) near models with 60 % stenosis. Importantly, SCE located from T1 to T5 exhibit a more pronounced reduction, exceeding 6.5, in the Womersley number. Our finding suggests that this region is the most vulnerable for SCE formation due to its significant impact on fluid circulation. The identification of specific locations within the SC associated with heightened risk can contribute to an improved understanding, treatment and management of SCE.
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Registered trials
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