Evidence map›Paper›PMID 39273026›Full record

ArticleCells2024

Modulation of Spontaneous Action Potential Rate by Inositol Trisphosphate in Myocytes from the Rabbit Atrioventricular Node.

Hongwei Cheng, Cherrie H T Kong, Andrew F James, Mark B Cannell, Jules C Hancox

Abstract read
In one paragraph

Article in Cells, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors.

Hongwei ChengSchool of Physiology, Pharmacology and Neuroscience, University of Bristol, Biomedical Sciences Building, University Walk, Bristol BS8 1TD, UK.
Cherrie H T KongSchool of Physiology, Pharmacology and Neuroscience, University of Bristol, Biomedical Sciences Building, University Walk, Bristol BS8 1TD, UK.
Andrew F JamesSchool of Physiology, Pharmacology and Neuroscience, University of Bristol, Biomedical Sciences Building, University Walk, Bristol BS8 1TD, UK.ORCID 0000-0002-2035-9489
Mark B CannellSchool of Physiology, Pharmacology and Neuroscience, University of Bristol, Biomedical Sciences Building, University Walk, Bristol BS8 1TD, UK.ORCID 0000-0002-4816-0463
Jules C HancoxSchool of Physiology, Pharmacology and Neuroscience, University of Bristol, Biomedical Sciences Building, University Walk, Bristol BS8 1TD, UK.

Funding

British Heart Foundation PG/14/42; PG/20/5/34801; PG/23/11356; FS/IBSRF/24/25203British Heart Foundation PG/20/5/34801
6 · The paper itself

Abstract

The atrioventricular node (AVN) is a key component of the cardiac conduction system and takes over pacemaking of the ventricles if the sinoatrial node fails. IP

Indexed as

Action PotentialsAtrioventricular NodeInositol 1,4,5-TrisphosphateInositol 1,4,5-Trisphosphate ReceptorsMyocytes, CardiacAnimalsMaleOxazolesRabbitsRyanodine Receptor Calcium Release ChannelInositol 1,4,5-TrisphosphateInositol 1,4,5-Trisphosphate ReceptorsOxazolesRyanodine Receptor Calcium Release Channel2-APBaction potentialatrioventricular nodeAVNIP3IP3-RIP3-R2pacemakingxestospongin C

Identifiers

PMID39273026
PMCPMC11394215

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.