ArticlePsychophysiology2024
Acute stress-induced reductions in neural response to reward are related to acute stress-related increases in cortisol.
Article in Psychophysiology, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
1 citing paper in PubMed.
- Influence of the HPA Axis on Anxiety-Related Processes: An RDoC Overview Considering Their Neural Correlates.Current psychiatry reports · 2025Review
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Authors and funding
4 authors.
Funding
Abstract
Stressors and blunted reward processing are implicated in depression. The current study simultaneously examined the impact of an acute stressor on cortisol and reward processing, measured using the reward positivity (RewP) in 66 participants. Participants completed a reward task during a stressor and a control condition, counterbalanced, and separated by 1 week, while saliva samples were collected before, immediately following, and 25 min after the reward task. Participants reported that the stressor condition was more stressful than the control condition. Cortisol levels did not differ before the reward task; however, cortisol levels were higher both immediately and 25 min after the task. The RewP was blunted during the stressor compared to the control condition, and participants with a larger stress-induced increase in cortisol had greater reductions in their RewP. The current study provides evidence that stress-induced changes in HPA-axis functioning relate to reductions in neural correlates of reward processing.
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Registered trials
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