Evidence map›Paper›PMID 39266539›Full record

ArticleCell death & disease2024

CD36-mediated ferroptosis destabilizes CD4

Hui Li, Peng-Fei Wang, Wei Luo, Di Fu, Wei-Yun Shen, Yan-Ling Zhang, Shuai Zhao, Ru-Ping Dai

Abstract read
In one paragraph

Article in Cell death & disease, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 17 papers.

0numbers the graph read from it
0cells of the map it votes in
17citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

17 citing papers in PubMed.

  1. Review
  2. Stress Hyperglycemia Drives CD4Advanced science (Weinheim, Baden-Wurttemberg, Germany) · 2026
    Article
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  6. Article
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  12. Article
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  16. Article
  17. [Research progress on the role of ferroptosis in aortic dissection].Zhejiang da xue xue bao. Yi xue ban = Journal of Zhejiang University. Medical sciences · 2024
    Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors.

Hui Li *Department of Anesthesiology, the Second XiangYa Hospital, Central South University, ChangSha, China.
Peng-Fei Wang *Department of Anesthesiology, the Second XiangYa Hospital, Central South University, ChangSha, China.
Wei LuoDepartment of Anesthesiology, the Second XiangYa Hospital, Central South University, ChangSha, China.
Di FuDepartment of Anesthesiology, XiangYa Hospital, Central South University, ChangSha, China.
Wei-Yun ShenDepartment of Anesthesiology, the Second XiangYa Hospital, Central South University, ChangSha, China.
Yan-Ling ZhangDepartment of Anesthesiology, the Second XiangYa Hospital, Central South University, ChangSha, China.
Shuai ZhaoDepartment of Anesthesiology, the Second XiangYa Hospital, Central South University, ChangSha, China. shuaizh730@csu.edu.cn.ORCID 0000-0001-7828-2915
Ru-Ping DaiDepartment of Anesthesiology, the Second XiangYa Hospital, Central South University, ChangSha, China. xyeyyrupingdai@csu.edu.cn.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Acute type A aortic dissection (ATAAD) is a lethal pathological process within the aorta with high mortality and morbidity. T lymphocytes are perturbed and implicated in the clinical outcome of ATAAD, but the exact characteristics of T cell phenotype and its underlying mechanisms in ATAAD remain poorly understood. Here we report that CD4

Indexed as

Aortic DissectionCD36 AntigensCD4-Positive T-LymphocytesFerroptosisHomeostasisAnimalsFemaleHumansMaleMiceMiddle AgedReactive Oxygen SpeciesCD36 AntigensCD36 protein, humanReactive Oxygen Species

Identifiers

PMID39266539
PMCPMC11392947

What OpenQuestion holds

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.