Evidence map›Paper›PMID 39256796›Full record

ArticleBMC biology2024

The m6A writer KIAA1429 regulates photoaging progression via MFAP4-dependent collagen synthesis.

Yuanyuan Liu, Jian Li, Chenhui Wang, Jiangbo Li, Kai Luo, Kang Tao, Yuan Tian, Xiang Song, Zhifang Zhai, Yuandong Tao and 6 more

Erratum issuedAbstract read
PubMed Full text at PMC Publisher
In one paragraph

Article in BMC biology, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. An erratum has been issued. Cited by 6 papers.

0numbers the graph read from it
0cells of the map it votes in
6citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

6 citing papers in PubMed.

  1. Article
  2. [Role of m6A methylation modification in photoaging and ultraviolet-induced skin cancers].Zhong nan da xue xue bao. Yi xue ban = Journal of Central South University. Medical sciences · 2025
    Review
  3. Article
  4. Article
  5. Review
  6. Article
4 · The record

Corrections and comments

5 · Who and what money

Authors and funding

16 authors.

Yuanyuan LiuMedical School of Chinese People's Liberation Army, Beijing, 100039, China.
Jian LiDepartment of Dermatology, Southwest Hospital, Army Medical University, Chongqing, 400038, China.
Chenhui Wang *Bioinformatics Center of AMMS, Beijing, 100063, China.
Jiangbo LiBioinformatics Center of AMMS, Beijing, 100063, China.
Kai LuoBiomedical Treatment Center, the Seventh Medical Center of Chinese, PLA General Hospital, Beijing, 100010, China.
Kang TaoDepartment of Dermatology, Southwest Hospital, Army Medical University, Chongqing, 400038, China.
Yuan TianDepartment of Dermatology, Southwest Hospital, Army Medical University, Chongqing, 400038, China.
Xiang SongBioinformatics Center of AMMS, Beijing, 100063, China.
Zhifang ZhaiDepartment of Dermatology, Southwest Hospital, Army Medical University, Chongqing, 400038, China.
Yuandong TaoDepartment of Pediatric Urology, the Seventh Medical Center of Chinese, PLA General Hospital, Beijing, 100010, China.
Jia YouBiomedical Treatment Center, the Seventh Medical Center of Chinese, PLA General Hospital, Beijing, 100010, China.
Lihua WuBiomedical Treatment Center, the Seventh Medical Center of Chinese, PLA General Hospital, Beijing, 100010, China.
Wenqian LiShandong University of Traditional Chinese Medicine, ShanDong, 250355, China.
Yuanyuan JiaoTianjin University of Traditional Chinese Medicine, Poyanghu Road, Tianjin, 301617, China.
Rongya YangDepartment of Dermatology, the Seventh Medical Center of Chinese PLA General Hospital, Beijing, 100010, China. RongyaYang960@outlook.com.
Mingwang ZhangDepartment of Dermatology, Southwest Hospital, Army Medical University, Chongqing, 400038, China. mingwangzhang56@outlook.com.

Funding

Chongqing Doctoral Through Train Research Project CSTB2022BSXM-JCX0021National Natural Science Foundation of China NO. 82002120National Natural Science Foundation of Chongqing NO. cstc2021jcyj-msxmX0140
6 · The paper itself

Abstract

backgroundN6-Methyladenosine (m

objectivesThis study aims to investigate the biological role and underlying mechanism of m

methodsm

resultsm

conclusionsThe increased expression of KIAA1429 hinders collagen synthesis during UVR-induced photoaging, suggesting that KIAA1429 represents a potential candidate for targeted therapy to mitigate UVR-driven photoaging.

Indexed as

CollagenSkin AgingAdenosineAnimalsFibroblastsHumansMethylationMiceUltraviolet RaysAdenosineCollagenN-methyladenosineKIAA1429MFAP4N6-methyladenosine (m6a)Photoaging

Identifiers

PMID39256796
PMCPMC11389345

What OpenQuestion holds

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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.