ArticleHeliyon2024
In-host density-dependent model of high-risk HPV virions, basal cells, lymphocytes t-cells incorporating functional responses.
Article in Heliyon, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Authors and funding
4 authors.
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Abstract
Cervical cancer is one of the most common types of cancer and it is caused mostly by high-risk Human Papillomavirus (HPV) and continues to spread at an alarming rate. While HPV impacts have been investigated before, there are currently only a scanty number of mathematical models that account for HPV's dynamic role in cervical cancer. The objectives were to develop an in-host density-dependent deterministic model for the dynamics implications of basal cells, virions, and lymphocytes incorporating immunity and functional responses. Analyze the model using techniques of epidemiological models such as basic reproduction number and simulate the model using Matlab ODE solver. Six compartments are considered in the model that is; Susceptible cells (S), Infected cells (I), Precancerous cells (P), Cancerous cells (C), Virions (V), and Lymphocytes (L). Next generation matrix (NGM), survival function, and characteristic polynomial method were used to determine the basic reproduction number denoted as
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