Evidence map›Paper›PMID 39183803›Full record

ArticleInfant and child development

Early risk for child externalising symptoms: Examining genetic, prenatal, temperamental and parental influences.

Sohee Lee, Olivia C Robertson, Kristine Marceau, Valerie S Knopik, Misaki Natsuaki, Daniel S Shaw, Leslie D Leve, Jody M Ganiban, Jenae M Neiderhiser

Abstract read
In one paragraph

Article in Infant and child development. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

9 authors.

Sohee LeeDepartment of Human Development and Family Science, Purdue University.
Olivia C RobertsonDepartment of Human Development and Family Science, Purdue University.
Kristine MarceauDepartment of Human Development and Family Science, Purdue University.
Valerie S KnopikDepartment of Human Development and Family Science, Purdue University.
Misaki NatsuakiDepartment of Psychology, University of California, Riverside.
Daniel S ShawDepartment of Psychology, University of Pittsburgh.
Leslie D LevePrevention Science Institute, University of Oregon.
Jody M GanibanDepartment of Psychology, George Washington University.
Jenae M NeiderhiserDepartment of Psychology, Penn State University.

Funding

The Early Growth and Development Study Pediatric CohortUH3OD023389 · OD · UNIVERSITY OF OREGON · PI Jody M. Ganiban, Leslie Diane Leve · 2018 to 2026
$16.6M
The Early Growth and Development Study: Family Process, Genes and School EntryR01HD042608 · NICHD · OREGON SOCIAL LEARNING CENTER, INC. · PI CAPALDI, DEBORAH M. · 2002 to 2011
$14.9M
Genes, Prenatal Drug Exposure, and the Postnatal Environment: An Adoption StudyR01DA020585 · NIDA · PENNSYLVANIA STATE UNIVERSITY, THE · PI NEIDERHISER, JENAE M. · 2007 to 2011
$5.9M
An adoption study of the development of early substance use: the joint roles of genetic influences, prenatal risk, rearing environment, and pubertal maturationR01DA045108 · NIDA · PENNSYLVANIA STATE UNIVERSITY, THE · PI NEIDERHISER, JENAE M. · 2018 to 2022
$3.7M
Gene-Environment Interplay and Development of Psychiatric Symptoms in ChildrenR01MH092118 · NIMH · PENNSYLVANIA STATE UNIVERSITY, THE · PI LEVE, LESLIE DIANE, NEIDERHISER, JENAE M. · 2010 to 2014
$3.1M
Gene-Environment interplay and childhood obesity: An Adoption study.R01DK090264 · NIDDK · GEORGE WASHINGTON UNIVERSITY · PI GANIBAN, JODY M. · 2011 to 2015
$2.8M
NICHD NIH HHS R01 HD042608NIDA NIH HHS R01 DA020585NIDA NIH HHS R01 DA045108NIDDK NIH HHS R01 DK090264NIH HHS UH3 OD023389NIMH NIH HHS R01 MH092118
6 · The paper itself

Abstract

This study utilized the Early Growth and Development Study (N = 561 adoptive children; 57.2% male, 55.3% White), a study of children adopted at birth, to examine heritable (birth parent psychopathology) and prenatal risk (prenatal maternal distress and smoking during pregnancy), infant negative affectivity, adoptive parent over-reactivity and warmth as independent predictors of childhood externalizing symptoms. The current study evaluated if: (1) infant negative affectivity and over-reactive parenting are candidate mediators for the effects of heritable and prenatal risk on externalizing symptoms and (2) parental warmth weakens the influence of heritable risk, prenatal risk, negative affectivity, and over-reactive parenting on externalizing symptoms. There were main effects of heritable risk, infant negative affectivity, and over-reactive parenting on child externalizing symptoms. The study found no support for the hypothesized mediation and moderation effects, suggesting that targeting parental over-reactivity rather than warmth would be more effective in reducing risk for childhood externalizing symptoms.

Indexed as

externalizing symptomsheritableover-reactive parentingparental warmthprenataltemperament

Identifiers

PMID39183803
PMCPMC11339911

What OpenQuestion holds

Textmetadata
LicenceTDM
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.