Evidence map›Paper›PMID 39170739›Full record

ReviewFrontiers in endocrinology2024

Metabolic alkalosis in cystic fibrosis: from vascular volume depletion to impaired bicarbonate excretion.

Manoocher Soleimani

Abstract readReview
In one paragraph

Review in Frontiers in endocrinology, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

1 author.

Manoocher SoleimaniDepartment of Medicine, University of New Mexico, Albuquerque, NM, United States.

Funding

The Role of Intercalated Cells and Their Acid Base and Electrolyte Transport Machinery in Kidney Cystogenesis by Tuberous SclerosisI01BX001000 · VA · CINCINNATI VA MEDICAL CENTER RESEARCH · PI SOLEIMANI, MANOOCHER · 2012 to 2025
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BLRD VA I01 BX001000
6 · The paper itself

Abstract

Cystic fibrosis (CF) is the most common life-threatening genetic disease in the United States and among people of European descent. Despite the widespread distribution of the cystic fibrosis transmembrane conductance regulator (CFTR) along kidney tubules, specific renal phenotypes attributable to CF have not been well documented. Recent studies have demonstrated the downregulation of the apical Cl

Indexed as

AlkalosisBicarbonatesCystic FibrosisSulfate TransportersAnimalsCystic Fibrosis Transmembrane Conductance RegulatorHumansMiceBicarbonatesCystic Fibrosis Transmembrane Conductance RegulatorSLC26A4 protein, humanSulfate Transportersacute kidney injuryBarter-like syndromediabetes mellitushypokalemiakidney tubulesnephrotoxicity

Identifiers

PMID39170739
PMCPMC11335532

What OpenQuestion holds

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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.