Evidence map›Paper›PMID 39135891›Full record

ArticleDiabetes, metabolic syndrome and obesity : targets and therapy2024

Association of Questionnaire-Based Physical Activity Analysis and Body Composition Dynamics in Type 2 Diabetes: A Cross-Sectional Study.

Dan-Dan Li, Xue Yang, Yang Yang, Li-Hua Zhao, Tian-Tian Zhang, Yi-Nan Wang, Jian-Bin Su, Li-Hua Wang, Hai-Yan Shi

Abstract read
In one paragraph

Article in Diabetes, metabolic syndrome and obesity : targets and therapy, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

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1 · What the graph read from it

What it found

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3 · Its place in the literature

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4 · The record

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5 · Who and what money

Authors and funding

9 authors.

Dan-Dan Li *Department of Endocrinology, Affiliated Hospital 2 of Nantong University, and First People's Hospital of Nantong City, Nantong, 226006, People's Republic of China.ORCID 0009-0003-9141-5382
Xue Yang *Department of Endocrinology, Affiliated Hospital 2 of Nantong University, and First People's Hospital of Nantong City, Nantong, 226006, People's Republic of China.
Yang Yang *Department of General Practice, Affiliated Hospital 2 of Nantong University, and First People's Hospital of Nantong City, Nantong, 226006, People's Republic of China.
Li-Hua ZhaoDepartment of Endocrinology, Affiliated Hospital 2 of Nantong University, and First People's Hospital of Nantong City, Nantong, 226006, People's Republic of China.
Tian-Tian ZhangDepartment of Hepatobiliary Surgery, Affiliated Hospital 2 of Nantong University, and First People's Hospital of Nantong City, Nantong, 226006, People's Republic of China.
Yi-Nan WangDepartment of Endocrinology, Affiliated Hospital 2 of Nantong University, and First People's Hospital of Nantong City, Nantong, 226006, People's Republic of China.
Jian-Bin SuDepartment of Endocrinology, Affiliated Hospital 2 of Nantong University, and First People's Hospital of Nantong City, Nantong, 226006, People's Republic of China.ORCID 0000-0002-2138-0263
Li-Hua WangDepartment of Nursing, Affiliated Hospital 2 of Nantong University, and First People's Hospital of Nantong City, Nantong, 226006, People's Republic of China.
Hai-Yan ShiDepartment of Hepatobiliary Surgery, Affiliated Hospital 2 of Nantong University, and First People's Hospital of Nantong City, Nantong, 226006, People's Republic of China.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Background: Physical activity (PA) exerts an important influence on glycemic control in type 2 diabetes (T2D) patients. Alterations in body composition in patients with T2D may be involved in the overall pathophysiologic process, but PAs and alterations in body composition have been poorly studied. Methods: A total of 615 patients with T2D were selected by convenient sampling. The patients were investigated with the International Physical Activity Questionnaire (IPAQ-S). Moreover, biochemical indices were collected, and the progression of the body composition of the subjects was determined via dual-energy X-ray absorptiometry (DXA). The variables included lumbar bone mineral density (LSBMD), femoral neck bone mineral density (FNBMD), hip bone mineral density (HBMD), whole-body bone mineral density (TBMD), limb skeletal muscle mass index (ASMI), whole-body fat percentage (B-FAT) and trunk fat percentage (T-FAT). Moreover, the levels of physical activity (high level of physical activity [H-PA], medium level of physical activity [M-PA] and low level of physical activity [L-PA]) were divided into three groups to analyze the changes in patient body composition with changes in physical activity level. Results: One-way analysis of variance showed that β-CTX, TP1NP, HbA1c, B-FAT and T-FAT increased significantly ( Conclusion: A lack of physical activity is an independent risk factor for decreased bone mineral density, decreased skeletal muscle content and increased fat content in patients with T2D.

Indexed as

body compositioncorrelationphysical activitytype 2 diabetes

Identifiers

PMID39135891
PMCPMC11318602

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