Evidence map›Paper›PMID 39120795›Full record

ArticleArchives of toxicology2024

Prolonged exposure to NaAsO

Lili Fan, Qian Song, Ying Jin, Rui He, Heng Diao, Peng Luo, Dapeng Wang

Abstract read
PubMed Publisher
In one paragraph

Article in Archives of toxicology, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.

0numbers the graph read from it
0cells of the map it votes in
3citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

3 citing papers in PubMed.

  1. Article
  2. Review
  3. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors.

Lili FanSchool of Public Health, Key Laboratory of Environmental Pollution Monitoring and Disease Control, Ministry of Education, Guizhou Medical University, Guiyang, 550025, Guizhou, People's Republic of China.
Qian SongSchool of Public Health, Key Laboratory of Environmental Pollution Monitoring and Disease Control, Ministry of Education, Guizhou Medical University, Guiyang, 550025, Guizhou, People's Republic of China.
Ying JinSchool of Public Health, Key Laboratory of Environmental Pollution Monitoring and Disease Control, Ministry of Education, Guizhou Medical University, Guiyang, 550025, Guizhou, People's Republic of China.
Rui HeSchool of Public Health, Key Laboratory of Environmental Pollution Monitoring and Disease Control, Ministry of Education, Guizhou Medical University, Guiyang, 550025, Guizhou, People's Republic of China.
Heng DiaoSchool of Public Health, Key Laboratory of Environmental Pollution Monitoring and Disease Control, Ministry of Education, Guizhou Medical University, Guiyang, 550025, Guizhou, People's Republic of China.
Peng LuoSchool of Public Health, Key Laboratory of Environmental Pollution Monitoring and Disease Control, Ministry of Education, Guizhou Medical University, Guiyang, 550025, Guizhou, People's Republic of China. luopeng@gmc.edu.cn.
Dapeng WangSchool of Public Health, Key Laboratory of Environmental Pollution Monitoring and Disease Control, Ministry of Education, Guizhou Medical University, Guiyang, 550025, Guizhou, People's Republic of China. wojiushiwdp@126.com.ORCID 0000-0001-5810-1806

Funding

Excellent Young Talents Plan of Guizhou Medical University [2023] No. 103Guizhou Provincial Science and Technology Department [2019] No. 1264Guizhou Provincial Science and Technology Department [2020] No. 4Y153National Natural Science Foundation of China 82060604Outstanding Young Scientific and Technological Talent Projects of Guizhou Province [2021] No. 5611
6 · The paper itself

Abstract

Arsenic, a well-known hazardous toxicant, has been found in recent years to act as an environmental endocrine disruptor that accumulates in various endocrine organs, impeding the normal physiological functions of these organs and altering hormone secretion levels. Moreover, some research has demonstrated a correlation between arsenic exposure and thyroid functions, suggesting that arsenic has a toxicological effect on the thyroid gland. However, the specific type of thyroid gland damage caused by arsenic exposure and its potential molecular mechanism remain poorly understood. In this study, the toxic effects of sodium arsenite (NaAsO

Indexed as

ArsenitesInflammasomesNLR Family, Pyrin Domain-Containing 3 ProteinPyroptosisRats, Sprague-DawleySodium CompoundsThyroid GlandAnimalsDose-Response Relationship, DrugEndocrine DisruptorsInflammationMaleRatsThyroid DiseasesThyroid HormonesArsenitesEndocrine DisruptorsInflammasomesNLR Family, Pyrin Domain-Containing 3 ProteinNlrp3 protein, ratsodium arseniteSodium CompoundsThyroid HormonesArsenicNLRP3 inflammasomePyroptosisRatsThyrotoxicity

Identifiers

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.