Evidence map›Paper›PMID 39117624›Full record

ArticleNature communications2024

Metabolic and transcriptomic reprogramming during contact inhibition-induced quiescence is mediated by YAP-dependent and YAP-independent mechanisms.

Soeun Kang, Maciek R Antoniewicz, Nissim Hay

Abstract read
In one paragraph

Article in Nature communications, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 9 papers.

0numbers the graph read from it
0cells of the map it votes in
9citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

9 citing papers in PubMed.

  1. Article
  2. Article
  3. Article
  4. Review
  5. Article
  6. Article
  7. The dynamics of centromere assembly and disassembly during quiescence.bioRxiv : the preprint server for biology · 2025
    Article
  8. Review
  9. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors.

Soeun KangDepartment of Biochemistry and Molecular Genetics, College of Medicine, University of Illinois at Chicago, Chicago, IL, USA.ORCID 0000-0002-0291-9542
Maciek R AntoniewiczChemical Engineering Department, University of Michigan, Ann Arbor, MI, USA.
Nissim HayDepartment of Biochemistry and Molecular Genetics, College of Medicine, University of Illinois at Chicago, Chicago, IL, USA. nhay@uic.edu.ORCID 0000-0002-6245-3000

Funding

PI3K/PTEN/Akt signaling and the genesis of cancerR01CA090764 · NCI · UNIVERSITY OF ILLINOIS AT CHICAGO · PI Nissim Hay · 2001 to 2026
$9.4M
The Role of Akt in Cell Survival and Cell GrowthR01AG016927 · NIA · UNIVERSITY OF ILLINOIS AT CHICAGO · PI HAY, NISSIM · 1998 to 2023
$9.0M
Hexokinase 2 and cancer therapyR01CA258299 · NCI · UNIVERSITY OF ILLINOIS AT CHICAGO · PI HAY, NISSIM · 2021 to 2025
$2.7M
Hexokinase 2 in liver cancerR01CA206167 · NCI · UNIVERSITY OF ILLINOIS AT CHICAGO · PI HAY, NISSIM · 2016 to 2020
$2.1M
Research Career Scientist AwardIK6BX004602 · VA · JESSE BROWN VA MEDICAL CENTER · PI Nissim Hay · 2019 to 2026
–
Hexokinase 2 and cancer therapyI01BX000733 · VA · JESSE BROWN VA MEDICAL CENTER · PI HAY, NISSIM · 2012 to 2020
–
The role of AMPK and CD36 in breast cancer tumorigenesis and metastasisI01BX005092 · VA · JESSE BROWN VA MEDICAL CENTER · PI HAY, NISSIM · 2021 to 2025
–
BLRD VA I01 BX000733BLRD VA I01 BX005092BLRD VA IK6 BX004602NCI NIH HHS R01 CA090764NCI NIH HHS R01 CA206167NCI NIH HHS R01 CA258299NIA NIH HHS R01 AG016927
6 · The paper itself

Abstract

Metabolic rewiring during the proliferation-to-quiescence transition is poorly understood. Here, using a model of contact inhibition-induced quiescence, we conducted

Indexed as

Adaptor Proteins, Signal TransducingCitric Acid CycleContact InhibitionFibroblastsGlycolysisMalate DehydrogenaseMitochondriaTranscriptomeYAP-Signaling ProteinsAnimalsCell Cycle ProteinsCell ProliferationMalatesMicePhosphoproteinsPyruvic AcidAdaptor Proteins, Signal TransducingCell Cycle ProteinsMalate DehydrogenaseMalatesmalic acidPhosphoproteinsPyruvic AcidYap1 protein, mouseYAP-Signaling Proteins

Identifiers

PMID39117624
PMCPMC11310444

What OpenQuestion holds

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.