Evidence map›Paper›PMID 39087342›Full record

ReviewCell proliferation2024

Comprehensive macro and micro views on immune cells in ischemic heart disease.

Yongjian Zhao, Mingyue Tan, Yunfei Yin, Jun Zhang, Yiyi Song, Hang Li, Lin Yan, Yifeng Jin, Ziyue Wu, Tianke Yang and 2 more

Abstract readReview
In one paragraph

Review in Cell proliferation, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 9 papers.

0numbers the graph read from it
0cells of the map it votes in
9citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

9 citing papers in PubMed.

  1. Article
  2. Review
  3. Review
  4. Review
  5. Article
  6. Article
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  9. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

12 authors.

Yongjian ZhaoDepartment of Cardiology, The First Affiliated Hospital of Soochow University, Suzhou, Jiangsu, China.
Mingyue TanDepartment of Cardiology, The First Affiliated Hospital of Soochow University, Suzhou, Jiangsu, China.
Yunfei YinDepartment of Cardiology, The First Affiliated Hospital of Soochow University, Suzhou, Jiangsu, China.
Jun ZhangDepartment of Cardiology, The First Affiliated Hospital of Soochow University, Suzhou, Jiangsu, China.
Yiyi SongSuzhou Medical College of Soochow University, Jiangsu, China.
Hang LiDepartment of Cardiology, The First Affiliated Hospital of Soochow University, Suzhou, Jiangsu, China.
Lin YanDepartment of Cardiology, The First Affiliated Hospital of Soochow University, Suzhou, Jiangsu, China.
Yifeng JinDepartment of Cardiology, The First Affiliated Hospital of Soochow University, Suzhou, Jiangsu, China.
Ziyue WuDepartment of Cardiology, The First Affiliated Hospital of Soochow University, Suzhou, Jiangsu, China.
Tianke YangDepartment of Ophthalmology, The First Affiliated Hospital of USTC, University of Science and Technology of China, Hefei, China.
Tingbo JiangDepartment of Cardiology, The First Affiliated Hospital of Soochow University, Suzhou, Jiangsu, China.
Hongxia LiDepartment of Cardiology, The First Affiliated Hospital of Soochow University, Suzhou, Jiangsu, China.ORCID https://orcid.org/0000-0002-1440-1345

Funding

National Natural Science Foundation of China 81100173National Natural Science Foundation of China 81770327Natural Science Foundation of Jiangsu Province BK20231198The Medical Research Project of Jiangsu Province Health Commission M2022038The Multi-center Clinical Research Project for Major Diseases in Suzhou DZXYJ202302
6 · The paper itself

Abstract

Ischemic heart disease (IHD) is a prevalent cardiovascular condition that remains the primary cause of death due to its adverse ventricular remodelling and pathological changes in end-stage heart failure. As a complex pathologic condition, it involves intricate regulatory processes at the cellular and molecular levels. The immune system and cardiovascular system are closely interconnected, with immune cells playing a crucial role in maintaining cardiac health and influencing disease progression. Consequently, alterations in the cardiac microenvironment are influenced and controlled by various immune cells, such as macrophages, neutrophils, dendritic cells, eosinophils, and T-lymphocytes, along with the cytokines they produce. Furthermore, studies have revealed that Gata6

Indexed as

Myocardial IschemiaAnimalsDendritic CellsHumansMacrophagesNeutrophilsT-Lymphocytes

Identifiers

PMID39087342
PMCPMC11628753

What OpenQuestion holds

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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.