Evidence map›Paper›PMID 39086825›Full record

ArticleRoyal Society open science2024

Mitochondrial function is enhanced by thyroid hormones during zebra finch development.

Marlene Oefele, Michaela Hau, Suvi Ruuskanen, Stefania Casagrande

Abstract read
In one paragraph

Article in Royal Society open science, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.

0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

2 citing papers in PubMed.

  1. Article
  2. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors.

Marlene OefeleEvolutionary Physiology Research Group, Max Planck Institute for Biological Intelligence, Eberhard-Gwinner-Strasse, Seewiesen 82319, Germany.ORCID https://orcid.org/0009-0008-4152-6320
Michaela HauEvolutionary Physiology Research Group, Max Planck Institute for Biological Intelligence, Eberhard-Gwinner-Strasse, Seewiesen 82319, Germany.
Suvi RuuskanenEnvironmental Physiology Research Group, University of Jyväskylä, Seminaarinkatu 15, University of Jyväskylä, Jyväskylä FI-40014, Finland.
Stefania CasagrandeEvolutionary Physiology Research Group, Max Planck Institute for Biological Intelligence, Eberhard-Gwinner-Strasse, Seewiesen 82319, Germany.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

An organism's response to its environment is largely determined by changes in the energy supplied by aerobic mitochondrial metabolism via adenosine triphosphate (ATP) production. ATP is especially important under energy-demanding conditions, such as during rapid growth. It is currently poorly understood how environmental factors influence energy metabolism and mitochondrial functioning, but recent studies suggest the role of thyroid hormones (TH). TH are key regulators of growth and metabolism and can be flexibly adjusted to environmental conditions, such as environmental temperature or food availability. To test whether TH enhancement is causally linked to mitochondrial function and growth, we provided TH orally at physiological concentrations during the main growth phase in zebra finch (

Indexed as

cellular respirationgrowthmetabolismmitochondriathyroid hormones

Identifiers

PMID39086825
PMCPMC11288688

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.