In one paragraphArticle in American journal of physiology. Heart and circulatory physiology, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
2 · The registryThe trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
3 · Its place in the literatureWho cites it
0 citing papers in PubMed.
No citing paper in PubMed yet.
4 · The recordCorrections and comments
5 · Who and what moneyAuthors and funding
11 authors.
Yara Bernaldo de QuirósDepartment of Integrative Physiology, University of Colorado Boulder, Boulder, Colorado, United States.ORCID 0000-0002-2611-0406 Sophia A MahoneyDepartment of Integrative Physiology, University of Colorado Boulder, Boulder, Colorado, United States.ORCID 0000-0002-0013-4514 Nicholas S VanDongenDepartment of Integrative Physiology, University of Colorado Boulder, Boulder, Colorado, United States.
Nathan T GreenbergDepartment of Integrative Physiology, University of Colorado Boulder, Boulder, Colorado, United States.ORCID 0000-0001-5174-3195 Ravinandan VenkatasubramanianDepartment of Integrative Physiology, University of Colorado Boulder, Boulder, Colorado, United States.
Pedro SaavedraDepartment of Mathematics, Universidad de Las Palmas de Gran Canaria, Las Palmas, Spain.ORCID 0000-0003-1681-7165 Greg BossartGeorgia Aquarium, Atlanta, Georgia, United States.
Vienna E BruntDepartment of Integrative Physiology, University of Colorado Boulder, Boulder, Colorado, United States.ORCID 0000-0002-1941-7936 Zachary S ClaytonDepartment of Integrative Physiology, University of Colorado Boulder, Boulder, Colorado, United States.ORCID 0000-0003-3878-3533 Antonio FernándezInstitute of Animal Health and Food Safety, Universidad de Las Palmas de Gran Canaria, Las Palmas, Spain.
Douglas R SealsDepartment of Integrative Physiology, University of Colorado Boulder, Boulder, Colorado, United States.ORCID 0000-0002-2391-9824 Funding
Translational studies of cellular senescence as a regulator of doxorubicin-mediated arterial dysfunctionR00HL159241 · NHLBI · UNIVERSITY OF COLORADO DENVER · PI Zachary S. Clayton · 2024 to 2026
$747kTranslational Studies of the Short-Chain Fatty Acid Acetate for Improving Age-Associated Arterial DysfunctionR00HL151818 · NHLBI · UNIVERSITY OF COLORADO DENVER · PI BRUNT, VIENNA E · 2022 to 2024
$747kTargeting cellular senescence to prevent accelerated vascular aging induced by the common chemotherapeutic agent doxorubicinR21AG078408 · NIA · UNIVERSITY OF COLORADO · PI SEALS, DOUGLAS R · 2022 to 2023
$430kTranslational studies of cellular senescence as a regulator of doxorubicin-mediated arterial dysfunctionK99HL159241 · NHLBI · UNIVERSITY OF COLORADO · PI CLAYTON, ZACHARY S. · 2022 to 2023
$350kTranslational Studies of the Short-Chain Fatty Acid Acetate for Improving Age-Associated Arterial DysfunctionK99HL151818 · NHLBI · UNIVERSITY OF COLORADO · PI BRUNT, VIENNA E · 2021 to 2022
$349kTargeting cellular senescence with oral fisetin supplementation to improve vascular agingF31HL165885 · NHLBI · UNIVERSITY OF COLORADO · PI MAHONEY, SOPHIA ANDREA · 2022 to 2025
$123kRole of the human gut microbiome in modulating age-associated aortic stiffening.F31HL164004 · NHLBI · UNIVERSITY OF COLORADO · PI GREENBERG, NATHAN THOMAS · 2022 to 2023
$70kEuropean Commission (EC) MSCA-IF-2019: 892267HHS | National Institutes of Health (NIH) F31: HL164004HHS | National Institutes of Health (NIH) F31: HL165885HHS | National Institutes of Health (NIH) K99/R00: HL151818HHS | National Institutes of Health (NIH) K99/R00: HL159241HHS | National Institutes of Health (NIH) R21: AG078408NHLBI NIH HHS F31 HL164004NHLBI NIH HHS F31 HL165885NHLBI NIH HHS K99 HL151818NHLBI NIH HHS K99 HL159241NHLBI NIH HHS R00 HL151818NHLBI NIH HHS R00 HL159241NIA NIH HHS R21 AG078408
6 · The paper itselfAbstract
Endothelial function declines with aging and independently predicts future cardiovascular disease (CVD) events. Diving also impairs endothelial function in humans. Yet, dolphins, being long-lived mammals adapted to diving, undergo repetitive cycles of tissue hypoxia-reoxygenation and disturbed shear stress without manifesting any apparent detrimental effects, as CVD is essentially nonexistent in these animals. Thus, dolphins may be a unique model of healthy arterial aging and may provide insights into strategies for clinical medicine. Emerging evidence shows that the circulating milieu (bioactive factors in the blood) is at least partially responsible for transducing reductions in age-related endothelial function. To assess whether dolphins have preserved endothelial function with aging because of a protected circulating milieu, we tested if the serum (pool of the circulating milieu) of bottlenose dolphins (
Indexed as
Bottle-Nosed DolphinEndothelium, VascularVasodilationAge FactorsAgingAnimalsArteriesFemaleHealthy AgingHumansMaleMice, Inbred C57BLModels, AnimalVasodilator AgentsVasodilator Agentsadvancing agecardiovascular diseasecetaceansdivingendothelial function
Identifiers
PMID39058431
PMCPMC11442021
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