Evidence map›Paper›PMID 39044930›Full record

ArticleJournal of medicine and life2024

Angiotensin II-independent abnormal renal vascular reactivity during puromycin nephropathy.

Luis Isaias Juncos, Akinwunmi Oluwaseun Adeoye, Fernando Luis Martin, Julio Pedro Juncos, Sandra Teresita Baigorria, Néstor Horacio García

Abstract read
In one paragraph

Article in Journal of medicine and life, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors.

Luis Isaias JuncosDepartment of Renal Physiology, J. Robert Cade Foundation, Córdoba, Argentina.
Akinwunmi Oluwaseun AdeoyeDepartment of Biochemistry, Federal University Oye-Ekiti, Ekiti state, Nigeria.
Fernando Luis MartinDepartment of Renal Physiology, J. Robert Cade Foundation, Córdoba, Argentina.
Julio Pedro JuncosDepartment of Renal Physiology, J. Robert Cade Foundation, Córdoba, Argentina.
Sandra Teresita BaigorriaDepartment of Renal Physiology, J. Robert Cade Foundation, Córdoba, Argentina.
Néstor Horacio GarcíaDepartment of Renal Physiology, J. Robert Cade Foundation, Córdoba, Argentina.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Experimental glomerulonephritis results in hypertension that is sensitive to salt. Nevertheless, salt retention alone cannot explain the increase in blood pressure. Angiotensin antagonistic therapy reduces hypertension caused by puromycin amino nucleosides (PAN). We investigated the hypothesis that PAN modifies renal vascular reactivity through processes dependent on angiotensin. Long-Evans rats were given an intraperitoneal injection of either puromycin (150 mg/kg) or saline (controls). Group 1 was fed a normal sodium diet (NSD,

Indexed as

Angiotensin IIKidneyAcetylcholineAnimalsBlood PressureKidney DiseasesMaleNitroprussidePuromycinPuromycin AminonucleosideRatsRats, Long-EvansAcetylcholineAngiotensin IINitroprussidePuromycinPuromycin Aminonucleosideglomerulonephritiskidneypuromycin nephropathyquinaprilsodium

Identifiers

PMID39044930
PMCPMC11262600

What OpenQuestion holds

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.