Evidence map›Paper›PMID 39031193›Full record

ArticleJournal of neurology2024

Novel variants in CSF1R associated with adult-onset leukoencephalopathy with axonal spheroids and pigmented glia (ALSP).

Anne S Schmitz, Janani Raju, Wolfgang Köhler, Stephan Klebe, Khaled Cheheb, Franziska Reschke, Saskia Biskup, Tobias B Haack, Benjamin Roeben, Melanie Kellner and 7 more

Abstract read
In one paragraph

Article in Journal of neurology, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 6 papers.

0numbers the graph read from it
0cells of the map it votes in
6citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

6 citing papers in PubMed.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

17 authors.

Anne S SchmitzHertie Institute for Clinical Brain Research, Tübingen, Germany.
Janani RajuHertie Institute for Clinical Brain Research, Tübingen, Germany.
Wolfgang KöhlerDepartment of Neurology, University Hospital Leipzig, Leipzig, Germany.
Stephan KlebeDepartment of Neurology, University Hospital Essen, Essen, Germany.
Khaled ChehebDepartment of Neurology, DRK Kamillus Klinik, Asbach, Germany.
Franziska ReschkeInstitute of Human Genetics, University of Leipzig Medical Center, Leipzig, Germany.
Saskia BiskupCeGaT GmbH and Zentrum Für Humangenetik, Tübingen, Germany.
Tobias B HaackInstitute of Medical Genetics and Applied Genomics, University Hospital Tübingen, Tübingen, Germany.
Benjamin RoebenHertie Institute for Clinical Brain Research, Tübingen, Germany.
Melanie KellnerHertie Institute for Clinical Brain Research, Tübingen, Germany.
Nils RahnerInstitut Für Klinische Genetik Und Tumorgenetik Bonn, Bonn, Germany.
Thomas BlochRadiologie Am Rhein, Bad Honnef, Germany.
Johannes LemkeInstitute of Human Genetics, University of Leipzig Medical Center, Leipzig, Germany.
Benjamin BenderDepartment of Neuroradiology, University Hospital Tübingen, Tübingen, Germany.
Ludger SchölsHertie Institute for Clinical Brain Research, Tübingen, Germany.
Holger HengelHertie Institute for Clinical Brain Research, Tübingen, Germany.
Stefanie N HayerHertie Institute for Clinical Brain Research, Tübingen, Germany. stefanie.hayer@googlemail.com.ORCID http://orcid.org/0000-0001-6944-6711

Funding

Bundesministerium für Gesundheit 2520DAT94BBundesministerium für Gesundheit ZMVI1-2520DAT94EGemeinnützige Hertie-Stiftung P1200021Medizinischen Fakultät, Eberhard Karls Universität Tübingen 478-0-0
6 · The paper itself

Abstract

The CSF1R gene, located on chromosome 5, encodes a 108 kDa protein and plays a critical role in regulating myeloid cell function. Mutations in CSF1R have been identified as a cause of a rare white matter disease called adult-onset leukoencephalopathy with axonal spheroids and pigmented glia (ALSP, also known as CSF1R-related leukoencephalopathy), characterized by progressive neurological dysfunction. This study aimed to broaden the genetic basis of ALSP by identifying novel CSF1R variants in patients with characteristic clinical and imaging features of ALSP. Genetic analysis was performed through whole-exome sequencing or panel analysis for leukodystrophy genes. Variant annotation and classification were conducted using computational tools, and the identified variants were categorized following the recommendations of the American College of Medical Genetics and Genomics (ACMG). To assess the evolutionary conservation of the novel variants within the CSF1R protein, amino acid sequences were compared across different species. The study identified six previously unreported CSF1R variants (c.2384G>T, c.2133_2919del, c.1837G>A, c.2304C>A, c.2517G>T, c.2642C>T) in seven patients with ALSP, contributing to the expanding knowledge of the genetic diversity underlying this rare disease. The analysis revealed considerable genetic and clinical heterogeneity among these patients. The findings emphasize the need for a comprehensive understanding of the genetic basis of rare diseases like ALSP and underscored the importance of genetic testing, even in cases with no family history of the disease. The study's contribution to the growing spectrum of ALSP genetics and phenotypes enhances our knowledge of this condition, which can be crucial for both diagnosis and potential future treatments.

Indexed as

LeukoencephalopathiesReceptors, Granulocyte-Macrophage Colony-Stimulating FactorAdultAgedFemaleHumansMaleMiddle AgedMutationNeurogliaReceptor, Macrophage Colony-Stimulating FactorCSF1R protein, humanReceptor, Macrophage Colony-Stimulating FactorReceptors, Granulocyte-Macrophage Colony-Stimulating FactorALSPCSF1RCSF1R-related leukoencephalopathyGenetic diagnostics

Identifiers

PMID39031193
PMCPMC11377666

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.