ArticleNature immunology2024
Reciprocal regulation of T follicular helper cells and dendritic cells drives colitis development.
Abstract read
In one paragraph
Article in Nature immunology, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 15 papers.
0numbers the graph read from it
0cells of the map it votes in
15citing papers in PubMed
–field-weighted citation impact
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
15 citing papers in PubMed.
- Cellular crosstalk of fibroblast-myofibroblast transition in intestinal homeostasis and disease.Biomarker research · 2026Review
- Targeting the immune-metabolic axis reverses orthodontics-induced systemic pathology.Signal transduction and targeted therapy · 2026Article
- A unique CD4⁺ T cell subset expressing granzyme K is regulated by transcription factor EOMES and important for T cell-mediated intestinal inflammation.Nature immunology · 2026Article
- Paeoniflorin as a Promising Therapeutic Candidate in Intestinal Diseases: Experimental Evidence, Mechanistic Insights, and Translational Perspectives.Drug design, development and therapy · 2026Review
- Follicular T cells in the ovarian cancer immune microenvironment: biological insights and translational implications.Frontiers in immunology · 2026Review
- The interaction between dendritic cells and T follicular helper cells drives inflammatory bowel disease: a review.Frontiers in immunology · 2026Review
- Fructose intake driven glycolysis-ROS-EGFR axis specifically promotes the generation and pathogenicity of Th17 cells.Nature communications · 2025Article
- Crosstalk between the microbiota and intestinal dendritic cells in IBD.Seminars in immunopathology · 2025Review
- Beyond T-cell subsets: stemness and adaptation redefining immunity and immunotherapy.Cellular & molecular immunology · 2025Review
- Smooth Muscle Silent Information Regulator 1 Contributes to Colitis in Mice.International journal of molecular sciences · 2025Article
- ALKBH5 acts a tumor-suppressive biomarker and is associated with immunotherapy response in hepatocellular carcinoma.Scientific reports · 2025Article
- Identification and Validation of Feature Genes Related to Mitochondrial Dysfunction and Oxidative Stress in Ulcerative Colitis.Journal of inflammation research · 2025Article
- Development of a mitochondria-related gene signature for prognostic assessment in diffuse large B cell lymphoma.Frontiers in oncology · 2025Article
- Gut Microbes Secretions May Trigger Mononuclear Cell Migration and Offer Comorbidity Mechanism Between Inflammatory Bowel Disease and Diabetic Retinopathy.Mediators of inflammation · 2025Article
- The role of dendritic cells in tertiary lymphoid structures: implications in cancer and autoimmune diseases.Frontiers in immunology · 2024Review
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
21 authors.
Xue BaiNew Cornerstone Science Laboratory, Shanghai Immune Therapy Institute, Shanghai Jiao Tong University School of Medicine-Affiliated Renji Hospital, Shanghai, China.ORCID http://orcid.org/0000-0002-9059-3078
Sijie ChenBioinformatics Division, BNRIST and Department of Automation, MOE Key Laboratory of Bioinformatics, Tsinghua University, Beijing, China.ORCID http://orcid.org/0000-0003-4331-0773
Xinxin ChiInstitute of Immunology and School of Medicine, Tsinghua University, Beijing, China.ORCID http://orcid.org/0000-0003-2120-6684
Bowen XieInstitute of Immunology and School of Medicine, Tsinghua University, Beijing, China.
Xinyi GuoNew Cornerstone Science Laboratory, Shanghai Immune Therapy Institute, Shanghai Jiao Tong University School of Medicine-Affiliated Renji Hospital, Shanghai, China.
Han FengInstitute of Immunology and School of Medicine, Tsinghua University, Beijing, China.
Peng WeiInstitute of Immunology and School of Medicine, Tsinghua University, Beijing, China.
Di ZhangDepartment of Pathology, The First Hospital of China Medical University and College of Basic Medical Sciences of China Medical University, Shenyang, China.
Shan XieDepartment of Gastroenterology, Beijing Tsinghua Changgung Hospital, School of Clinical Medicine, Tsinghua University, Beijing, China.
Tian XieInstitute of Immunology and School of Medicine, Tsinghua University, Beijing, China.
Yongzhen ChenInstitute of Immunology and School of Medicine, Tsinghua University, Beijing, China.
Mengting GouNew Cornerstone Science Laboratory, Shanghai Immune Therapy Institute, Shanghai Jiao Tong University School of Medicine-Affiliated Renji Hospital, Shanghai, China.
Qin QiaoInstitute of Immunology and School of Medicine, Tsinghua University, Beijing, China.
Xinwei LiuInstitute of Immunology and School of Medicine, Tsinghua University, Beijing, China.
Wei JinInstitute of Immunology and School of Medicine, Tsinghua University, Beijing, China.
Wei XuInstitute of Immunology and School of Medicine, Tsinghua University, Beijing, China.
Zixuan ZhaoInstitute of Immunology and School of Medicine, Tsinghua University, Beijing, China.
Qi XingNew Cornerstone Science Laboratory, Shanghai Immune Therapy Institute, Shanghai Jiao Tong University School of Medicine-Affiliated Renji Hospital, Shanghai, China.
Xiaohu WangInstitute of Immunology and School of Medicine, Tsinghua University, Beijing, China.
Xuegong ZhangBioinformatics Division, BNRIST and Department of Automation, MOE Key Laboratory of Bioinformatics, Tsinghua University, Beijing, China.ORCID http://orcid.org/0000-0002-9684-5643
Chen DongNew Cornerstone Science Laboratory, Shanghai Immune Therapy Institute, Shanghai Jiao Tong University School of Medicine-Affiliated Renji Hospital, Shanghai, China. dongchen@westlake.edu.cn.ORCID http://orcid.org/0000-0002-0084-9130
Funding
National Natural Science Foundation of China (National Science Foundation of China) 31500743National Natural Science Foundation of China (National Science Foundation of China) 31821003National Natural Science Foundation of China (National Science Foundation of China) 32070889National Natural Science Foundation of China (National Science Foundation of China) 32370938National Natural Science Foundation of China (National Science Foundation of China) 91642201National Natural Science Foundation of China (National Science Foundation of China) 92374001Shanghai Science and Technology Development Foundation (Shanghai Science and Technology Development Fund) 21JC1404200
Abstract
The immunological mechanisms underlying chronic colitis are poorly understood. T follicular helper (T
Indexed as
Cell DifferentiationColitisDendritic CellsT Follicular Helper CellsAnimalsColonDisease Models, AnimalGerminal CenterMiceMice, Inbred C57BLMice, KnockoutMice, TransgenicTh1 Cells
Identifiers
PMID
38942990What OpenQuestion holds
Textmetadata
Read underepoch 390
Registered trials
None linked
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.