Evidence map›Paper›PMID 38935536›Full record

ReviewClinical and translational medicine2024

Harnessing CD8

Bing Yue, Yuxia Gao, Yi Hu, Meixiao Zhan, Yangzhe Wu, Ligong Lu

Abstract readReview
In one paragraph

Review in Clinical and translational medicine, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 19 papers.

0numbers the graph read from it
0cells of the map it votes in
19citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

19 citing papers in PubMed.

  1. Review
  2. Article
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  8. Article
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  10. Article
  11. Article
  12. Article
  13. Review
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  15. Review
  16. Article
  17. Review
  18. Manipulating the 4-1BB Pathway to Cure HBV.Infectious diseases & immunity · 2024
    Article
  19. Harnessing CD8Clinical and translational medicine · 2024
    Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors.

Bing YueGuangdong Provincial Key Laboratory of Tumour Interventional Diagnosis and Treatment, Zhuhai Institute of Translational Medicine, Zhuhai Clinical Medical College of Jinan University (Zhuhai People's Hospital), Jinan University, Zhuhai, Guangdong, China.
Yuxia GaoGuangdong Provincial Key Laboratory of Tumour Interventional Diagnosis and Treatment, Zhuhai Institute of Translational Medicine, Zhuhai Clinical Medical College of Jinan University (Zhuhai People's Hospital), Jinan University, Zhuhai, Guangdong, China.
Yi HuMicrobiology and Immunology Department, School of Medicine, Faculty of Medical Science, Jinan University, Guangzhou, Guangdong, China.
Meixiao ZhanGuangdong Provincial Key Laboratory of Tumour Interventional Diagnosis and Treatment, Zhuhai Institute of Translational Medicine, Zhuhai Clinical Medical College of Jinan University (Zhuhai People's Hospital), Jinan University, Zhuhai, Guangdong, China.
Yangzhe WuGuangdong Provincial Key Laboratory of Tumour Interventional Diagnosis and Treatment, Zhuhai Institute of Translational Medicine, Zhuhai Clinical Medical College of Jinan University (Zhuhai People's Hospital), Jinan University, Zhuhai, Guangdong, China.ORCID 0000-0002-5270-2747
Ligong LuGuangdong Provincial Key Laboratory of Tumour Interventional Diagnosis and Treatment, Zhuhai Institute of Translational Medicine, Zhuhai Clinical Medical College of Jinan University (Zhuhai People's Hospital), Jinan University, Zhuhai, Guangdong, China.

Funding

National Natural Science Foundation of China 32030036National Natural Science Foundation of China 32270950National Natural Science Foundation of China 82002787National Natural Science Foundation of China 82230067Natural Science Foundation of Guangdong Province of China 2414050004764Startup Foundation of the Zhuhai People's Hospital YNXM20210305
6 · The paper itself

Abstract

Hepatitis B virus (HBV) infection playsa significant role in the etiology and progression of liver-relatedpathologies, encompassing chronic hepatitis, fibrosis, cirrhosis, and eventual hepatocellularcarcinoma (HCC). Notably, HBV infection stands as the primary etiologicalfactor driving the development of HCC. Given the significant contribution ofHBV infection to liver diseases, a comprehensive understanding of immunedynamics in the liver microenvironment, spanning chronic HBV infection,fibrosis, cirrhosis, and HCC, is essential. In this review, we focused on thefunctional alterations of CD8

Indexed as

CD8-Positive T-LymphocytesHepatitis B virusCarcinoma, HepatocellularHepatitis B, ChronicHumansLiver DiseasesLiver NeoplasmsCD8+ T Cell DynamicsHBV‐Associated Liver DiseasesReview

Identifiers

PMID38935536
PMCPMC11210506

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.