Evidence map›Paper›PMID 38924413›Full record

ArticleScience advances2024

H3K9 methylation regulates heterochromatin silencing through incoherent feedforward loops.

Kannosuke Yabe, Asuka Kamio, Satoyo Oya, Tetsuji Kakutani, Mami Hirayama, Yuriko Tanaka, Soichi Inagaki

Abstract read
In one paragraph

Article in Science advances, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 7 papers.

0numbers the graph read from it
0cells of the map it votes in
7citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

7 citing papers in PubMed.

  1. Review
  2. Review
  3. Article
  4. Lactate Enhances CD8International journal of molecular sciences · 2026
    Article
  5. Article
  6. Article
  7. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors.

Kannosuke YabeDepartment of Biological Sciences, Graduate School of Science, The University of Tokyo, Tokyo, Japan.ORCID 0009-0001-8844-6896
Asuka KamioDepartment of Biological Sciences, Graduate School of Science, The University of Tokyo, Tokyo, Japan.
Satoyo OyaDepartment of Biological Sciences, Graduate School of Science, The University of Tokyo, Tokyo, Japan.ORCID 0000-0002-8019-1665
Tetsuji KakutaniDepartment of Biological Sciences, Graduate School of Science, The University of Tokyo, Tokyo, Japan.ORCID 0000-0002-6137-4474
Mami HirayamaDepartment of Biological Sciences, Graduate School of Science, The University of Tokyo, Tokyo, Japan.
Yuriko TanakaDepartment of Biological Sciences, Graduate School of Science, The University of Tokyo, Tokyo, Japan.
Soichi InagakiDepartment of Biological Sciences, Graduate School of Science, The University of Tokyo, Tokyo, Japan.ORCID 0000-0003-4090-826X

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Histone H3 lysine-9 methylation (H3K9me) is a hallmark of the condensed and transcriptionally silent heterochromatin. It remains unclear how H3K9me controls transcription silencing and how cells delimit H3K9me domains to avoid silencing essential genes. Here, using

Indexed as

ArabidopsisArabidopsis ProteinsGene SilencingHeterochromatinHistonesEpigenesis, GeneticGene Expression Regulation, PlantHistone-Lysine N-MethyltransferaseLysineMethylationArabidopsis ProteinsHeterochromatinHistone-Lysine N-MethyltransferaseHistonesLysine

Identifiers

PMID38924413
PMCPMC11204290

What OpenQuestion holds

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LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.