Evidence map›Paper›PMID 38892395›Full record

ReviewInternational journal of molecular sciences2024

Animal Models and Molecular Pathogenesis of Arrhythmogenic Cardiomyopathy Associated with Pathogenic Variants in Intercalated Disc Genes.

Sara Vencato, Chiara Romanato, Alessandra Rampazzo, Martina Calore

Abstract readReview
In one paragraph

Review in International journal of molecular sciences, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 4 papers.

0numbers the graph read from it
0cells of the map it votes in
4citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

4 citing papers in PubMed.

  1. Article
  2. Article
  3. Review
  4. Gene Therapy TargetingMedComm · 2025
    Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors.

Sara VencatoDepartment of Biology, University of Padova, Via U. Bassi 58/B, 35121 Padova, Italy.
Chiara RomanatoDepartment of Biology, University of Padova, Via U. Bassi 58/B, 35121 Padova, Italy.
Alessandra RampazzoDepartment of Biology, University of Padova, Via U. Bassi 58/B, 35121 Padova, Italy.ORCID 0000-0003-3884-8843
Martina CaloreDepartment of Biology, University of Padova, Via U. Bassi 58/B, 35121 Padova, Italy.

Funding

European Commission 101115536
6 · The paper itself

Abstract

Arrhythmogenic cardiomyopathy (ACM) is a rare genetic cardiac disease characterized by the progressive substitution of myocardium with fibro-fatty tissue. Clinically, ACM shows wide variability among patients; symptoms can include syncope and ventricular tachycardia but also sudden death, with the latter often being its sole manifestation. Approximately half of ACM patients have been found with variations in one or more genes encoding cardiac intercalated discs proteins; the most involved genes are plakophilin 2 (

Indexed as

Arrhythmogenic Right Ventricular DysplasiaDisease Models, AnimalAnimalsDesmoglein 2DesmoplakinsDesmosomesHumansMicePlakophilinsWnt Signaling PathwayDesmoglein 2DesmoplakinsPlakophilinsarrhythmogenic cardiomyopathyintercalated discmurine modelspathogenic mechanisms

Identifiers

PMID38892395
PMCPMC11172742

What OpenQuestion holds

Textmetadata
LicenceCC BY
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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.