Evidence map›Paper›PMID 38883815›Full record

ArticleiScience2024

TRIM41 contributes to the pathogenesis of airway allergy by compromising dendritic cells' tolerogenic properties.

Qiuying Peng, Xiangqian Luo, Lihua Mo, Xuejie Xu, Yu Liu, Dabo Liu, Pingchang Yang

Abstract read
PubMed Full text at PMC Publisher
In one paragraph

Article in iScience, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors.

Qiuying PengDepartment of Pediatric Otolaryngology, Shenzhen Hospital of Southern Medical University, Shenzhen, China.
Xiangqian LuoDepartment of Pediatric Otolaryngology, Shenzhen Hospital of Southern Medical University, Shenzhen, China.
Lihua MoDepartment of Pediatric Otolaryngology, Shenzhen Hospital of Southern Medical University, Shenzhen, China.
Xuejie XuInstitute of Allergy & Immunology of Shenzhen University and State Key Laboratory of Respiratory Diseases Allergy Division at Shenzhen University, Shenzhen, China.
Yu LiuDepartment of General Practice Medicine, Third Affiliated Hospital of Shenzhen University, Shenzhen, China.
Dabo LiuDepartment of Pediatric Otolaryngology, Shenzhen Hospital of Southern Medical University, Shenzhen, China.
Pingchang YangInstitute of Allergy & Immunology of Shenzhen University and State Key Laboratory of Respiratory Diseases Allergy Division at Shenzhen University, Shenzhen, China.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Dendritic cells (DC) play a crucial role in the initiation of immune responses. TRIM41, an E3 ubiquitin ligase, can facilitate targeting protein degradation. The purpose of this study is to analyze the role of TRIM41 in the pathogenesis of airway allergy (AA) and the impact of regulating TRIM41 on suppressing AA. We observed that the airway DCs of AA mice had a higher expression of

Indexed as

Components of the immune systemImmunologyMolecular biology

Identifiers

PMID38883815
PMCPMC11176661

What OpenQuestion holds

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.