Evidence map›Paper›PMID 38883785›Full record

ArticleResearch square2024

Single-cell transcriptomic analyses of mouse idh1 mutant growth plate chondrocytes reveal distinct cell populations responsible for longitudinal growth and enchondroma formation.

Vijitha Puviindran, Eijiro Shimada, Zeyu Huang, Xinyi Ma, Ga I Ban, Yu Xiang, Hongyuan Zhang, Jianhong Ou, Xiaolin Wei, Makoto Nakagawa and 3 more

Abstract readPreprint
In one paragraph

Article in Research square, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

5 · Who and what money

Authors and funding

13 authors.

Vijitha PuviindranDuke University School of Medicine.
Eijiro ShimadaDuke University School of Medicine.
Zeyu HuangDuke University School of Medicine.
Xinyi MaDuke University School of Medicine.
Ga I BanDuke University School of Medicine.
Yu XiangDuke University School of Medicine.
Hongyuan ZhangDuke University School of Medicine.
Jianhong OuDuke University School of Medicine.
Xiaolin WeiDuke University School of Medicine.
Makoto NakagawaDuke University School of Medicine.
John MartinDuke University School of Medicine.
Yarui DiaoDuke University School of Medicine.
Benjamin A AlmanDuke University School of Medicine.

Funding

Molecular Etiology of Enchondromatosis: Hedgehog Regulation in ChondrocytesR01AR066765 · NIAMS · DUKE UNIVERSITY · PI ALMAN, BENJAMIN AARON · 2014 to 2025
$3.7M
Exercise-Induced Recovery of Intervertebral Disc HealthR00AR077685 · NIAMS · RUSH UNIVERSITY MEDICAL CENTER · PI MARTIN, JOHN · 2023 to 2025
$732k
NIAMS NIH HHS R00 AR077685NIAMS NIH HHS R01 AR066765
6 · The paper itself

Abstract

Enchondromas are a common tumor in bone that can occur as multiple lesions in enchondromatosis, which is associated with deformity of the effected bone. These lesions harbor mutations in

Identifiers

PMID38883785
PMCPMC11178001

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.