Evidence map›Paper›PMID 38791389›Full record

ReviewInternational journal of molecular sciences2024

Human and Murine Toll-like Receptor-Driven Disease in Systemic Lupus Erythematosus.

Susannah von Hofsten, Kristin Andreassen Fenton, Hege Lynum Pedersen

Abstract readReview
In one paragraph

Review in International journal of molecular sciences, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 17 papers.

0numbers the graph read from it
0cells of the map it votes in
17citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

17 citing papers in PubMed.

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  15. Immune Mechanisms and Biomarkers in Systemic Lupus Erythematosus.International journal of molecular sciences · 2024
    Article
  16. Review
  17. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors.

Susannah von HofstenDepartment of Medical Biology, Faculty of Health Sciences, UiT The Arctic University of Norway, 9019 Tromsø, Norway.ORCID 0000-0002-4079-0655
Kristin Andreassen FentonCentre of Clinical Research and Education, University Hospital of North Norway, Department of Medical Biology, Faculty of Health Sciences, UiT The Arctic University of Norway, 9019 Tromsø, Norway.ORCID 0000-0002-8356-2628
Hege Lynum PedersenCentre of Clinical Research and Education, University Hospital of North Norway, Department of Medical Biology, Faculty of Health Sciences, UiT The Arctic University of Norway, 9019 Tromsø, Norway.ORCID 0000-0002-3768-9513

Funding

Northern Norway Regional Health Authority HNF1343-17Northern Norway Regional Health Authority HNF1427-18
6 · The paper itself

Abstract

The pathogenesis of systemic lupus erythematosus (SLE) is linked to the differential roles of toll-like receptors (TLRs), particularly TLR7, TLR8, and TLR9. TLR7 overexpression or gene duplication, as seen with the Y-linked autoimmune accelerator (

Indexed as

Lupus Erythematosus, SystemicToll-Like Receptor 7AnimalsDisease Models, AnimalGenetic Predisposition to DiseaseHumansMiceToll-Like Receptor 8Toll-Like ReceptorsTLR7 protein, humanToll-Like Receptor 7Toll-Like Receptor 8Toll-Like Receptorsmouse modelssystemic lupus erythematosustoll-like receptor

Identifiers

PMID38791389
PMCPMC11120885

What OpenQuestion holds

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Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.