Evidence map›Paper›PMID 38791293›Full record

ArticleInternational journal of molecular sciences2024

Homogalacturonan Pectins Tuned as an Effect of Susceptible rbohD, Col-0-Reactions, and Resistance rbohF-, rbohD/F-Reactions to TuMV.

Katarzyna Otulak-Kozieł, Edmund Kozieł, Krzysztof Treder, Piotr Rusin

Abstract read
In one paragraph

Article in International journal of molecular sciences, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.

0numbers the graph read from it
0cells of the map it votes in
3citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

3 citing papers in PubMed.

  1. Review
  2. Review
  3. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors.

Katarzyna Otulak-KoziełDepartment of Botany, Institute of Biology, Warsaw University of Life Sciences-SGGW, Nowoursynowska Street 159, 02-776 Warsaw, Poland.ORCID 0000-0001-9424-1662
Edmund KoziełDepartment of Botany, Institute of Biology, Warsaw University of Life Sciences-SGGW, Nowoursynowska Street 159, 02-776 Warsaw, Poland.ORCID 0000-0001-6646-1254
Krzysztof TrederPlant Breeding and Acclimatization Institute-National Research Institute in Radzików, Bonin Division, Department of Potato Protection and Seed Science at Bonin, Bonin Str. 3, 76-009 Bonin, Poland.ORCID 0000-0002-8764-0574
Piotr RusinDepartment of Botany, Institute of Biology, Warsaw University of Life Sciences-SGGW, Nowoursynowska Street 159, 02-776 Warsaw, Poland.

Funding

National Science Center NCN 2021/43/D/NZ3/00428
6 · The paper itself

Abstract

The plant cell wall is an actively reorganized network during plant growth and triggered immunity in response to biotic stress. While the molecular mechanisms managing perception, recognition, and signal transduction in response to pathogens are well studied in the context of damaging intruders, the current understanding of plant cell wall rebuilding and active defense strategies in response to plant virus infections remains poorly characterized. Pectins can act as major elements of the primary cell wall and are dynamic compounds in response to pathogens. Homogalacturonans (HGs), a main component of pectins, have been postulated as defensive molecules in plant-pathogen interactions and linked to resistance responses. This research focused on examining the regulation of selected pectin metabolism components in susceptible (

Indexed as

ArabidopsisCell WallDisease ResistancePectinsPlant DiseasesArabidopsis ProteinsCarboxylic Ester HydrolasesGene Expression Regulation, PlantPotyvirusArabidopsis ProteinsCarboxylic Ester HydrolasespectinesterasePectinspolygalacturonic acidcell wall changespectinplant-virus interactionsultrastructure

Identifiers

PMID38791293
PMCPMC11120978

What OpenQuestion holds

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.