Evidence map›Paper›PMID 38782543›Full record

ArticleOpen heart2024

Association between speckle tracking echocardiography and pressure-volume loops during cardiogenic shock development.

Peter Hartmund Frederiksen, Louise Linde, Emilie Gregers, Nanna Louise Junker Udesen, Ole K Helgestad, Ann Banke, Jordi Sanchez Dahl, Amalie L Povlsen, Lisette Okkels Jensen, Jeppe P Larsen and 4 more

Abstract read
In one paragraph

Article in Open heart, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed, 1 pooled it
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

2 citing papers in PubMed, 1 synthesis or guideline pooled it.

  1. Pooled it
  2. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

14 authors.

Peter Hartmund FrederiksenDepartment of Cardiology, Odense University Hospital, Odense, Denmark peter.hartmund.frederiksen@rsyd.dk.ORCID 0000-0003-3712-7980
Louise LindeDepartment of Cardiology, Odense University Hospital, Odense, Denmark.
Emilie GregersDepartment of Cardiology, Copenhagen University Hospital, Kobenhavn, Denmark.
Nanna Louise Junker UdesenDepartment of Cardiology, Odense University Hospital, Odense, Denmark.
Ole K HelgestadDepartment of Cardiology, Odense University Hospital, Odense, Denmark.
Ann BankeDepartment of Cardiology, Odense University Hospital, Odense, Denmark.
Jordi Sanchez DahlDepartment of Cardiology, Odense University Hospital, Odense, Denmark.
Amalie L PovlsenDepartment of Cardiothoracic Anaesthesiology, Odense University Hospital, Odense, Denmark.
Lisette Okkels JensenDepartment of Cardiology, Odense University Hospital, Odense, Denmark.
Jeppe P LarsenDepartment of Cardiothoracic Anaesthesiology, Odense University Hospital, Odense, Denmark.
Jens LassenDepartment of Cardiology, Odense University Hospital, Odense, Denmark.
Henrik SchmidtDepartment of Cardiothoracic Anaesthesiology, Odense University Hospital, Odense, Denmark.
Hanne Berg Ravn *University of Southern Denmark, Odense, Denmark.
Jacob Eifer Moller *Department of Cardiology, Odense University Hospital, Odense, Denmark.ORCID 0000-0003-2873-5845

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

backgroundThe relationship between speckle tracking assessed global longitudinal strain (GLS) and Doppler-based echocardiography with basic physiological markers of cardiac function derived from pressure-volume loops is poorly elucidated.

objectiveWe aimed to describe the association between LS and Doppler-based echocardiography and direct measurements of central haemodynamic parameters from conductance catheter-based pressure-volume loops in an animal model with increasing left ventricular (LV) dysfunction.

methods12 Danish landrace female pigs (75-80 kg) were used. All instrumentations were performed percutaneously, including the conductance catheter in the LV. Progressive LV dysfunction was induced by embolisation through the left main coronary artery with microspheres every 3 min until a >50% reduction in cardiac output (CO) or mixed venous saturation (SvO

resultsWith progressive LV dysfunction, mean CO decreased from 5.6±0.9 L/min to 2.1±0.9 L/min, and mean SvO

conclusionIn an animal model of acute progressive LV dysfunction, echocardiographic and conductance catheter-based measurements changed significantly. LS and LVOT VTI displayed the earliest and the largest alterations with increased myocardial damage and both correlated strongest with stroke work.

Indexed as

Disease Models, AnimalShock, CardiogenicVentricular Dysfunction, LeftVentricular Function, LeftAnimalsEchocardiography, DopplerFemalePredictive Value of TestsSwineAcute Coronary SyndromeDisease Models, AnimalEchocardiographyHeart Failure

Identifiers

PMID38782543
PMCPMC11116883

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.