Evidence map›Paper›PMID 38765941›Full record

ArticlePsychiatry and clinical psychopharmacology2021

Association of the Uncoupling Protein 2-866 G/A Polymorphism with Family History and Duration of Tobacco Use Disorder in a Turkish Population.

Hasan Mervan Aytac, Sacide Pehlivan, Selin Kurnaz, Mustafa Pehlivan, Pinar Cetinay Aydin

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Article in Psychiatry and clinical psychopharmacology, 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

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0citing papers in PubMed
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1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

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Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors.

Hasan Mervan AytacDepartment of Psychiatry, Basaksehir Cam and Sakura City Hospital, İstanbul, Turkey.
Sacide PehlivanDepartment of Medical Biology, Istanbul University School of Medicine, İstanbul, Turkey.
Selin KurnazDepartment of Medical Biology, Istanbul University School of Medicine, İstanbul, Turkey.
Mustafa PehlivanDepartment of Internal Medicine Division of Hematology, Gaziantep University, Gaziantep, Turkey.
Pinar Cetinay AydinDepartment of Psychiatry, Psychiatry Clinic, Bakirkoy Research and Training Hospital for Psychiatry, Neurology and Neurosurgery, University of Health Sciences, Istanbul, Turkey.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Background: A variety of substances cause neurotoxicity by increasing intracellular oxidative stress, followed by mitochondrial dysfunction. Uncoupling proteins (UCPs) act as membrane transport proteins and reduce reactive oxygen products and mitochondrial calcium influx. We aimed to study Methods: One hundred eighteen patients with TUD and 96 healthy volunteers were included in the study. The diagnosis of the patients were then confirmed, based on the DSM-5 criteria. Polymerase chain reaction and restriction fragment length polymorphism (PCR-RFLP) were used to determine Results: Our results demonstrated that the Conclusions: In summary, the

Indexed as

Nicotinesingle nucleotide polymorphismtobacco use disorderUCP2-866 G/Auncoupling protein 2

Identifiers

PMID38765941
PMCPMC11079660

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.