Evidence map›Paper›PMID 38742227›Full record

ReviewFrontiers in molecular neuroscience2024

The role of

Taku Ito, Hiroki Watanabe, Keiji Honda, Taro Fujikawa, Ken Kitamura, Takeshi Tsutsumi

Abstract readReview
In one paragraph

Review in Frontiers in molecular neuroscience, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed.

  1. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors.

Taku ItoDepartment of Otorhinolaryngology, Tokyo Medical and Dental University, Tokyo, Japan.
Hiroki WatanabeDepartment of Otorhinolaryngology, Tokyo Medical and Dental University, Tokyo, Japan.
Keiji HondaDepartment of Otorhinolaryngology, Tokyo Medical and Dental University, Tokyo, Japan.
Taro FujikawaDepartment of Otorhinolaryngology, Tokyo Medical and Dental University, Tokyo, Japan.
Ken KitamuraDepartment of Otorhinolaryngology, Chigasaki Chuo Hospital, Kanagawa, Japan.
Takeshi TsutsumiDepartment of Otorhinolaryngology, Tokyo Medical and Dental University, Tokyo, Japan.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Inner ear malformations are predominantly attributed to developmental arrest during the embryonic stage of membranous labyrinth development. Due to the inherent difficulty in clinically assessing the status of the membranous labyrinth, these malformations are diagnosed with radiographic imaging, based on the morphological characteristics of the bony labyrinth. While extensive research has elucidated the intricacies of membranous labyrinth development in mouse models, comprehensive investigations into the developmental trajectory of the bony labyrinth, especially about its calcification process, have been notably lacking. One of the most prominent types of inner ear malformations is known as incomplete partition (IP), characterized by nearly normal external cochlear appearance but pronounced irregularities in the morphology of the modiolus and inter-scalar septa. IP type II (IP-II), also known as Mondini dysplasia, is generally accompanied by an enlargement of the vestibular aqueduct and is primarily attributed to mutations in the

Indexed as

bony labyrinthincomplete partition type II, SLC26A4micro-CTotic capsuleotoconia

Identifiers

PMID38742227
PMCPMC11089141

What OpenQuestion holds

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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.