Evidence map›Paper›PMID 38733386›Full record

ArticleJournal of molecular medicine (Berlin, Germany)2024

Blocking H

Baoling Zhu, Yi Yang, Xiangfei Wang, Dili Sun, Xiyang Yang, Xiaowei Zhu, Suling Ding, Chun Xiao, Yunzeng Zou, Xiangdong Yang

Abstract read
PubMed Publisher
In one paragraph

Article in Journal of molecular medicine (Berlin, Germany), 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 5 papers.

0numbers the graph read from it
0cells of the map it votes in
5citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

5 citing papers in PubMed.

  1. Article
  2. The role ofBlood science (Baltimore, Md.) · 2026
    Article
  3. Review
  4. Review
  5. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

10 authors.

Baoling Zhu *Shanghai Institute of Cardiovascular Diseases, Zhongshan Hospital & Institutes of Biomedical Sciences, Fudan University, Shanghai, 200032, China.
Yi Yang *Department of Medical Laboratory, College of Medical Technology, Shanghai University of Medicine & Health Sciences, Shanghai, 201318, China.
Xiangfei Wang *Department of Cardiology, Zhongshan Hospital, Fudan University, Shanghai, 200032, China.
Dili Sun *Department of Cardiology, Zhongshan Hospital, Fudan University, Shanghai, 200032, China.
Xiyang YangDepartment of Cardiology, Zhongshan Hospital, Fudan University, Shanghai, 200032, China.
Xiaowei ZhuDepartment of Cardiology, Zhongshan Hospital, Fudan University, Shanghai, 200032, China.
Suling DingShanghai Institute of Cardiovascular Diseases, Zhongshan Hospital & Institutes of Biomedical Sciences, Fudan University, Shanghai, 200032, China.
Chun XiaoDepartment of Cardiology, Third People's Hospital of Huizhou, Guangdong, 516003, China. huizhouxiaoc@163.com.
Yunzeng ZouShanghai Institute of Cardiovascular Diseases, Zhongshan Hospital & Institutes of Biomedical Sciences, Fudan University, Shanghai, 200032, China. zou.yunzeng@zs-hospital.sh.cn.
Xiangdong YangShanghai Institute of Cardiovascular Diseases, Zhongshan Hospital & Institutes of Biomedical Sciences, Fudan University, Shanghai, 200032, China. yangxiangdong_zs@163.com.ORCID 0000-0002-4201-7017

Funding

Guangdong Basic and Applied Basic Research Foundation 2022A1515140176Shanghai Sailing Program 23YF1431600the Key Project of the Science and Technology Committee of Baoshan district of Shanghai 21-E-42the National Natural Science Foundation of China 82170258the Science Foundation of Shanghai Committee of Science and Technology 22ZR1446700
6 · The paper itself

Abstract

Atherosclerosis (AS) is a chronic inflammatory arterial disease, in which abnormal lipid metabolism and foam cell formation play key roles. Histamine is a vital biogenic amine catalyzed by histidine decarboxylase (HDC) from L-histidine. Histamine H1 receptor (H

Indexed as

AtherosclerosisFoam CellsInflammationMice, KnockoutReceptors, Histamine H1Signal TransductionAnimalsApolipoproteins EDiet, High-FatDisease Models, AnimalLipid MetabolismMaleMiceMice, Inbred C57BLp38 Mitogen-Activated Protein KinasesPlaque, AtheroscleroticApolipoproteins Ep38 Mitogen-Activated Protein KinasesReceptors, Histamine H1AtherosclerosisH1RLIPGMacrophage-derived foam cell formationP38 MAPK

Identifiers

PMID38733386

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.