Evidence map›Paper›PMID 38725538›Full record

ArticleEpilepsy & behavior reports2024

Total callosotomy ameliorates epileptic activity and improves cognitive function in a patient with Miller-Dieker syndrome.

Masataka Fukuoka, Ichiro Kuki, Yuka Hattori, Hitomi Tsuji, Asako Horino, Megumi Nukui, Takeshi Inoue, Shin Okazaki, Noritsugu Kunihiro, Takehiro Uda

Abstract readCase Reports
In one paragraph

Article in Epilepsy & behavior reports, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.

0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

2 citing papers in PubMed.

  1. Article
  2. Understanding the Molecular Basis of Miller-Dieker Syndrome.International journal of molecular sciences · 2025
    Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

10 authors.

Masataka FukuokaDepartment of Pediatric Neurology, Osaka City General Hospital, Osaka, Japan.
Ichiro KukiDepartment of Pediatric Neurology, Osaka City General Hospital, Osaka, Japan.
Yuka HattoriDepartment of Pediatric Neurology, Osaka City General Hospital, Osaka, Japan.
Hitomi TsujiDepartment of Pediatric Neurology, Osaka City General Hospital, Osaka, Japan.
Asako HorinoDepartment of Pediatric Neurology, Osaka City General Hospital, Osaka, Japan.
Megumi NukuiDepartment of Pediatric Neurology, Osaka City General Hospital, Osaka, Japan.
Takeshi InoueDepartment of Pediatric Neurology, Osaka City General Hospital, Osaka, Japan.
Shin OkazakiDepartment of Pediatric Neurology, Osaka City General Hospital, Osaka, Japan.
Noritsugu KunihiroDepartment of Pediatric Neurosurgery, Osaka City General Hospital, Osaka, Japan.
Takehiro UdaDepartment of Pediatric Neurosurgery, Osaka City General Hospital, Osaka, Japan.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Miller-Dieker syndrome (MDS) is characterized by facial abnormalities and lissencephaly and is caused by a microdeletion in the region containing the

Indexed as

CallosotomyEpileptic spasmsLissencephalyMiller-Dieker syndrome

Identifiers

PMID38725538
PMCPMC11079450

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC-ND
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.