Evidence map›Paper›PMID 38700693›Full record

ArticleEuropean journal of pediatrics2024

Genotype and clinical phenotype of children with Marfan syndrome in Southeastern Anatolia.

Murat Karaoglan, Gulper Nacarkahya, Emel Hatun Aytac, Mehmet Keskin

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Article in European journal of pediatrics, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

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4 · The record

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5 · Who and what money

Authors and funding

4 authors.

Murat KaraoglanFaculty of Medicine, Department of Pediatric Endocrinology, Gaziantep University, Gaziantep, Turkey. muratkaraoglan@gantep.edu.tr.ORCID http://orcid.org/0000-0002-2861-3568
Gulper NacarkahyaDepartment of Molecular Biology, Gaziantep University Faculty of Medicine, Gaziantep, Turkey.ORCID https://orcid.org/0000-0002-8512-8833
Emel Hatun AytacFaculty of Medicine, Department of Pediatric Endocrinology, Gaziantep University, Gaziantep, Turkey.ORCID https://orcid.org/0000-0002-8385-4049
Mehmet KeskinFaculty of Medicine, Department of Pediatric Endocrinology, Gaziantep University, Gaziantep, Turkey.ORCID https://orcid.org/0000-0002-5544-8991

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

The cardinal phenotypic hallmarks of Marfan syndrome (MFS) include cardiac, ocular, and skeletal abnormalities. Since the clinical phenotype of MFS is highly heterogeneous, with certain symptoms appearing as children age, the diagnostic process and establishing a genotype-phenotype association in childhood MFS can be challenging. The lack of sufficient childhood studies also makes it difficult to interpret the subject. This study aims to evaluate the relationship between clinical symptoms used as diagnostic criteria and FBN1 variations in children with MFS. This study investigated the relationships between genotypes and phenotypes in 131 children suspected of having Marfan syndrome (MFS). Diagnosis of MFS was made according to the revised Ghent nosology. FBN1 variants were categorized based on exon regions, type of variant, and pathogenicity classes. These FBN1 variants were then correlated with the clinical manifestations including cardiovascular, ocular, facial, and skeletal abnormalities. Out of the children, 43 were diagnosed with MFS. FBN1 variant was identified in 32 (74.4%) of the MFS children. MFS diagnosis could not be made in five (15.6%) FBN1 variant-positive children. The most common cardinal finding is cardiac anomalies n = 38 (88.3%). The most common FBN1 pathogenic variant was c.1786 T > C/p.Cys596Arg n = 4 (12.5%). The distribution of pathogenic variants was as follows: 29 (90.6%) missense, 2 (6.3%) frameshift, and 1 (3.1%) nonsense. The numbers of AD and EL of the variant-positive children were 16 (50%) and 14 (43.7%), respectively. Ocular abnormalities were more common in children with FBN1-positive MFS (p = 0.009). There was no difference in the number of cardiac abnormalities between FBN1-positive and FBN1-negative MFS patients (p = 0.139).   Conclusion: This study examines the relationship between FBN1 variants and clinical features used as diagnostic criteria in MFS children. The findings emphasize the importance of long-term monitoring of heterogeneous clinical phenotypes and bioinformatic reanalysis in determining the genotype-phenotype relationship in children, as MFS symptoms can vary with age. What is Known: • Marfan syndrome has highly variable phenotypic heterogeneity. • The genotype-phenotype relationship in childhood Marfan syndrome is not clear enough due to the variation in the time of onset of the findings. What is New: • This article provides regional data for the field of research on genotype-phenotype relationships in childhood Marfan syndrome. • Long-term follow-up of clinical findings and bioinformatics reanalysis is an important requirement for a well-established genotype-phenotype relationship in childhood Marfan syndrome.

Indexed as

Fibrillin-1GenotypeMarfan SyndromePhenotypeAdipokinesAdolescentChildChild, PreschoolFemaleGenetic Association StudiesHumansInfantMaleMutationTurkeyAdipokinesFBN1 protein, humanFibrillin-1Aortic dilatationEctopia lentisFBN1Marfan syndrome

Identifiers

PMID38700693
PMCPMC11263224

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