Evidence map›Paper›PMID 38655005›Full record

ArticlePulmonary circulation2024

Early identification of SOX17 deficiency in infants to guide management of heritable pulmonary arterial hypertension using PDA stent to create reverse Potts shunt physiology.

Heidi Ostler, Carolyn Fall, Howaida El-Said, Henri Justino, Shylah Haldeman, Jeanne Carroll, Rohit Rao

Open access · goldAbstract readCase Reports
PubMed Full text at PMC Publisher
In one paragraph

Article in Pulmonary circulation, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.

0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed
1.7field-weighted citation impact, top 16% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

2 citing papers in PubMed, 4 citations in OpenAlex.

  1. Review
  2. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors at 2 institutions in 1 country.

Heidi OstlerDepartment of Pediatrics, Division of Pediatric Cardiology, Rady Children's Hospital University of California San Diego California USA.ORCID 0000-0003-4038-9850
Carolyn FallDepartment of Pediatrics, Division of Pediatric Cardiology, Rady Children's Hospital University of California San Diego California USA.
Howaida El-SaidDepartment of Pediatrics, Division of Pediatric Cardiology, Rady Children's Hospital University of California San Diego California USA.
Henri JustinoDepartment of Pediatrics, Division of Pediatric Cardiology, Rady Children's Hospital University of California San Diego California USA.
Shylah HaldemanDepartment of Pediatrics, Division of Pediatric Cardiology Rady Children's Hospital San Diego California USA.
Jeanne CarrollDepartment of Pediatrics, Division of Pediatric Cardiology, Rady Children's Hospital University of California San Diego California USA.
Rohit RaoDepartment of Pediatrics, Division of Pediatric Cardiology, Rady Children's Hospital University of California San Diego California USA.
University of California San Diego · USRady Children's Hospital-San Diego · US

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Heritable pulmonary arterial hypertension (HPAH) is a rare progressive condition that includes patients with an identified genetic cause of pulmonary arterial hypertension (PAH). HPAH and idiopathic PAH (IPAH) have an estimated combined incidence of 0.5-0.9 cases per million children-years. Several pathogenic variants have been associated with HPAH in children and adults, including genes

Indexed as

geneticspatent ductus arteriosuspediatric cardiovascular diseasepediatricspulmonary arterial hypertension

Identifiers

PMID38655005
PMCPMC11035901
OpenAlexW4395045116

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.